about
IL-2 reverses established type 1 diabetes in NOD mice by a local effect on pancreatic regulatory T cellsCutting edge: NF-κB p65 and c-Rel control epidermal development and immune homeostasis in the skinκB-Ras proteins regulate both NF-κB-dependent inflammation and Ral-dependent proliferation.Pathogenic T cells have a paradoxical protective effect in murine autoimmune diabetes by boosting Tregs.PDK1 Is a Regulator of Epidermal Differentiation that Activates and Organizes Asymmetric Cell Division.An NF-κB Transcription-Factor-Dependent Lineage-Specific Transcriptional Program Promotes Regulatory T Cell Identity and Function.NF-κB c-Rel Is Crucial for the Regulatory T Cell Immune Checkpoint in Cancer.The Alternative NF-κB Pathway in Regulatory T Cell Homeostasis and Suppressive Function.A Novel Link between Inflammation and Cancer.[Could we cure type 1 diabetes by stimulating T(reg)?]Effector T cells boost regulatory T cell expansion by IL-2, TNF, OX40, and plasmacytoid dendritic cells depending on the immune contextCutaneous p38 mitogen-activated protein kinase activation triggers psoriatic dermatitis
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P50
description
researcher ORCID ID = 0000-0002-3515-8305
@en
wetenschapper
@nl
name
Yenkel Grinberg-Bleyer
@ast
Yenkel Grinberg-Bleyer
@en
Yenkel Grinberg-Bleyer
@es
Yenkel Grinberg-Bleyer
@nl
type
label
Yenkel Grinberg-Bleyer
@ast
Yenkel Grinberg-Bleyer
@en
Yenkel Grinberg-Bleyer
@es
Yenkel Grinberg-Bleyer
@nl
prefLabel
Yenkel Grinberg-Bleyer
@ast
Yenkel Grinberg-Bleyer
@en
Yenkel Grinberg-Bleyer
@es
Yenkel Grinberg-Bleyer
@nl
P106
P31
P496
0000-0002-3515-8305