about
Secreted M-ficolin anchors onto monocyte transmembrane G protein-coupled receptor 43 and cross talks with plasma C-reactive protein to mediate immune signaling and regulate host defenseLocal inflammation induces complement crosstalk which amplifies the antimicrobial responseA computational and experimental study of the regulatory mechanisms of the complement systemHistidine-mediated pH-sensitive regulation of M-ficolin:GlcNAc binding activity in innate immunity examined by molecular dynamics simulations.Probiotic L. reuteri treatment prevents bone loss in a menopausal ovariectomized mouse model.Loss of Bone and Wnt10b Expression in Male Type 1 Diabetic Mice Is Blocked by the Probiotic Lactobacillus reuteri.Natural IgG antibodies provide innate protection against ficolin-opsonized bacteria.Molecular interaction between natural IgG and ficolin--mechanistic insights on adaptive-innate immune crosstalk.Both spontaneous Ins2(+/-) and streptozotocin-induced type I diabetes cause bone loss in young mice.The actin-related p41ARC subunit contributes to p21-activated kinase-1 (PAK1)-mediated glucose uptake into skeletal muscle cells.Doc2b Protects β-Cells Against Inflammatory Damage and Enhances Function.
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P50
description
researcher ORCID ID = 0000-0003-0391-7298
@en
name
Jing Zhang
@ast
Jing Zhang
@en
Jing Zhang
@es
Jing Zhang
@nl
type
label
Jing Zhang
@ast
Jing Zhang
@en
Jing Zhang
@es
Jing Zhang
@nl
prefLabel
Jing Zhang
@ast
Jing Zhang
@en
Jing Zhang
@es
Jing Zhang
@nl
P108
P106
P1960
MPGTgbEAAAAJ
P2456
05/3499-20
P31
P496
0000-0003-0391-7298