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Contribution of Hfe expression in macrophages to the regulation of hepatic hepcidin levels and iron loadingAvoiding transcription factor competition at promoter level increases the chances of obtaining oscillation.Neuroferritinopathy: missense mutation in FTL causing early-onset bilateral pallidal involvement.Contribution of STAT3 and SMAD4 pathways to the regulation of hepcidin by opposing stimuli.A biobrick library for cloning custom eukaryotic plasmids.Exclusion of mutations in the PRNP, JPH3, TBP, ATN1, CREBBP, POU3F2 and FTL genes as a cause of disease in Portuguese patients with a Huntington-like phenotype.Anemia upregulates lipocalin 2 in the liver and serum.Building blocks for protein interaction devices.Distinct requirements for Hfe in basal and induced hepcidin levels in iron overload and inflammation.Repression of repulsive guidance molecule C during inflammation is independent of Hfe and involves tumor necrosis factor-alphaHfe and Hjv exhibit overlapping functions for iron signaling to hepcidin.
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description
researcher ORCID ID = 0000-0002-7448-5552
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wetenschapper
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name
Marco Constante
@ast
Marco Constante
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Marco Constante
@es
Marco Constante
@nl
type
label
Marco Constante
@ast
Marco Constante
@en
Marco Constante
@es
Marco Constante
@nl
prefLabel
Marco Constante
@ast
Marco Constante
@en
Marco Constante
@es
Marco Constante
@nl
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0000-0002-7448-5552