about
Profile of gantenerumab and its potential in the treatment of Alzheimer's diseaseThe GTP-binding protein Rhes modulates dopamine signalling in striatal medium spiny neuronsRasd2 Modulates Prefronto-Striatal Phenotypes in Humans and 'Schizophrenia-Like Behaviors' in Mice.Dysfunctional dopaminergic neurotransmission in asocial BTBR mice.A role for D-aspartate oxidase in schizophrenia and in schizophrenia-related symptoms induced by phencyclidine in miceRhes influences striatal cAMP/PKA-dependent signaling and synaptic plasticity in a gender-sensitive fashion.A physiological mechanism to regulate D-aspartic acid and NMDA levels in mammals revealed by D-aspartate oxidase deficient mice.Free D-aspartate regulates neuronal dendritic morphology, synaptic plasticity, gray matter volume and brain activity in mammalsVoluntary exercise and sucrose consumption enhance cannabinoid CB1 receptor sensitivity in the striatum.Abnormal NMDA receptor function exacerbates experimental autoimmune encephalomyelitisD-Aspartate: An endogenous NMDA receptor agonist enriched in the developing brain with potential involvement in schizophrenia.Rhes, a striatal-enriched small G protein, mediates mTOR signaling and L-DOPA-induced dyskinesia.Age-Related Changes in D-Aspartate Oxidase Promoter Methylation Control Extracellular D-Aspartate Levels and Prevent Precocious Cell Death during Brain Aging.Dopamine D2 receptor dysfunction is rescued by adenosine A2A receptor antagonism in a model of DYT1 dystonia.D-aspartate modulates nociceptive-specific neuron activity and pain threshold in inflammatory and neuropathic pain condition in mice.Persistent increase of D-aspartate in D-aspartate oxidase mutant mice induces a precocious hippocampal age-dependent synaptic plasticity and spatial memory decay.Higher free D-aspartate and N-methyl-D-aspartate levels prevent striatal depotentiation and anticipate L-DOPA-induced dyskinesia.Role of aberrant striatal dopamine D1 receptor/cAMP/protein kinase A/DARPP32 signaling in the paradoxical calming effect of amphetamine.A fast and sensitive method for measuring picomole levels of total free amino acids in very small amounts of biological tissues.D-aspartate prevents corticostriatal long-term depression and attenuates schizophrenia-like symptoms induced by amphetamine and MK-801.D-aspartate dysregulation in Ddo(-/-) mice modulates phencyclidine-induced gene expression changes of postsynaptic density molecules in cortex and striatum.Increased D-aspartate brain content rescues hippocampal age-related synaptic plasticity deterioration of mice.d-Aspartate oxidase influences glutamatergic system homeostasis in mammalian brain.DNA methylation state of BDNF gene is not altered in prefrontal cortex and striatum of schizophrenia subjects.Decreased levels of D-aspartate and NMDA in the prefrontal cortex and striatum of patients with schizophrenia.
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description
onderzoeker
@nl
researcher ORCID ID = 0000-0002-3636-7395
@en
name
Francesco Errico
@ast
Francesco Errico
@en
Francesco Errico
@es
Francesco Errico
@nl
type
label
Francesco Errico
@ast
Francesco Errico
@en
Francesco Errico
@es
Francesco Errico
@nl
prefLabel
Francesco Errico
@ast
Francesco Errico
@en
Francesco Errico
@es
Francesco Errico
@nl
P106
P1153
23479578800
P21
P31
P496
0000-0002-3636-7395