about
Spontaneous bacterial peritonitis: The clinical challenge of a leaky gut and a cirrhotic liverLow-dose interleukin-2 promotes STAT-5 phosphorylation, Treg survival and CTLA-4-dependent function in autoimmune liver diseasesGenetic polymorphisms associated with fatty liver disease and fibrosis in HIV positive patients receiving combined antiretroviral therapy (cART).Influence of the CXCL1 rs4074 A allele on alcohol induced cirrhosis and HCC in patients of European descent.Impact of rifaximin on the frequency and characteristics of spontaneous bacterial peritonitis in patients with liver cirrhosis and ascitesAlterations of the NK cell pool in HIV/HCV co-infectionCompartment-specific distribution of human intestinal innate lymphoid cells is altered in HIV patients under effective therapy.IL-28B Genetic Variants Determine the Extent of Monocyte-Induced Activation of NK Cells in Hepatitis C.Relative Ascites Polymorphonuclear Cell Count Indicates Bacterascites and Risk of Spontaneous Bacterial Peritonitis.Influence of genetic variations in the SOD1 gene on the development of ascites and spontaneous bacterial peritonitis in decompensated liver cirrhosis.Hypoxia impairs anti-viral activity of natural killer (NK) cells but has little effect on anti-fibrotic NK cell functions in hepatitis C virus infection.A common polymorphism in the NCAN gene is associated with hepatocellular carcinoma in alcoholic liver disease.MicroRNA-155 is upregulated in ascites in patients with spontaneous bacterial peritonitis.Antibiotic resistance in healthcare-related and nosocomial spontaneous bacterial peritonitis.The ratio of calprotectin to total protein as a diagnostic and prognostic marker for spontaneous bacterial peritonitis in patients with liver cirrhosis and ascites.A variant in the nuclear dot protein 52kDa gene increases the risk for spontaneous bacterial peritonitis in patients with alcoholic liver cirrhosis.Regulatory CD4+ T cells modulate the interaction between NK cells and hepatic stellate cells by acting on either cell type.A farnesoid X receptor polymorphism predisposes to spontaneous bacterial peritonitis.Induction of stearoyl-CoA desaturase protects human arterial endothelial cells against lipotoxicity.Misery acquaints a man with dangerous bedfellows: But how to get rid of them?[A patient with striking skull and stomach][73-year-old patient with hepatocellular carcinoma]Low ascitic fluid protein does not indicate an increased risk for spontaneous bacterial peritonitis in current cohortsHow to reduce mortality of bacterascites-That is the question
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description
onderzoeker
@nl
researcher ORCID ID = 0000-0002-4921-200X
@en
name
Philipp Lutz
@ast
Philipp Lutz
@en
Philipp Lutz
@es
Philipp Lutz
@nl
type
label
Philipp Lutz
@ast
Philipp Lutz
@en
Philipp Lutz
@es
Philipp Lutz
@nl
prefLabel
Philipp Lutz
@ast
Philipp Lutz
@en
Philipp Lutz
@es
Philipp Lutz
@nl
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35786036300
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0000-0002-4921-200X