about
Immune-Mediated Inflammation May Contribute to the Pathogenesis of Cardiovascular Disease in Mucopolysaccharidosis Type IBrown fat activation reduces hypercholesterolaemia and protects from atherosclerosis development.Ext1 heterozygosity causes a modest effect on postprandial lipid clearance in humans.The plasma concentration of HDL-associated apoM is influenced by LDL receptor-mediated clearance of apoB-containing particles.Apolipoproteins E and AV mediate lipoprotein clearance by hepatic proteoglycansHepatic remnant lipoprotein clearance by heparan sulfate proteoglycans and low-density lipoprotein receptors depend on dietary conditions in miceSmall molecule antagonists of cell-surface heparan sulfate and heparin-protein interactions.ApoC-III inhibits clearance of triglyceride-rich lipoproteins through LDL family receptors.Opposing effects of apolipoprotein m on catabolism of apolipoprotein B-containing lipoproteins and atherosclerosis.Guanidinylated Neomycin Conjugation Enhances Intranasal Enzyme Replacement in the Brain.Human Cytomegalovirus Replication is Inhibited by the Autophagy-Inducing Compounds Trehalose and SMER28 Through Distinctively Different Mechanisms.Novel Lipoprotein(a) Catabolism Pathway via Apolipoprotein(a) Recycling: Adding the Plasminogen Receptor PlgRKT to the List.Dietary nitrate struggles in atherosclerosis.ANGPTL3 targeting: The power of versatile lipid-lowering.Heparan sulfate proteoglycans fine-tune macrophage inflammation via IFN-β.Human species-specific loss of CMP-N-acetylneuraminic acid hydroxylase enhances atherosclerosis via intrinsic and extrinsic mechanismsInactivation of the proximal NPXY motif impairs early steps in LRP1 biosynthesisThe heparan sulfate proteoglycan grip on hyperlipidemia and atherosclerosisThe adaptor protein PID1 regulates receptor-dependent endocytosis of postprandial triglyceride-rich lipoproteinsZNF263 is a transcriptional regulator of heparin and heparan sulfate biosynthesisHepatic heparan sulfate is a master regulator of hepcidin expression and iron homeostasis in human hepatocytes and miceReply to Soulillou et al.: Difficulties in extrapolating from animal models exemplify unusual human atherosclerosis susceptibility and mechanisms via CMAH loss
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P50
description
onderzoeker
@nl
researcher ORCID ID = 0000-0001-7224-4328
@en
name
Philip L Gordts
@ast
Philip L Gordts
@en
Philip L Gordts
@es
Philip L Gordts
@nl
type
label
Philip L Gordts
@ast
Philip L Gordts
@en
Philip L Gordts
@es
Philip L Gordts
@nl
prefLabel
Philip L Gordts
@ast
Philip L Gordts
@en
Philip L Gordts
@es
Philip L Gordts
@nl
P106
P1153
34771506800
P31
P496
0000-0001-7224-4328