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Trpm4 gene invalidation leads to cardiac hypertrophy and electrophysiological alterationsAccessory Kvbeta1 subunits differentially modulate the functional expression of voltage-gated K+ channels in mouse ventricular myocytesInitiation of embryonic cardiac pacemaker activity by inositol 1,4,5-trisphosphate-dependent calcium signalingA critical role for PPARalpha-mediated lipotoxicity in the pathogenesis of diabetic cardiomyopathy: modulation by dietary fat content.Cardiac specification of embryonic stem cells.Antagonism of Nav channels and α1-adrenergic receptors contributes to vascular smooth muscle effects of ranolazine.PPARalpha-mediated remodeling of repolarizing voltage-gated K+ (Kv) channels in a mouse model of metabolic cardiomyopathy.Role of heteromultimers in the generation of myocardial transient outward K+ currents.Biodegradable Polymeric Nanocapsules Prevent Cardiotoxicity of Anti-Trypanosomal Lychnopholide.Ionic basis of ventricular arrhythmias in remodeled rat heart during long-term myocardial infarction.The p42/44mitogen-activated protein kinase inhibitor PD 98059, but not U 0126, increases a K+ current in cardiomyocytes.Functional remodeling in post-myocardial infarcted rats: focus on beta-adrenoceptor subtypes.The TRPM4 channel is functionally important for the beneficial cardiac remodeling induced by endurance training.Functional evidence for an active role of B-type natriuretic peptide in cardiac remodelling and pro-arrhythmogenicity.Microarray analysis reveals complex remodeling of cardiac ion channel expression with altered thyroid status: relation to cellular and integrated electrophysiology.Akt regulates L-type Ca2+ channel activity by modulating Cavα1 protein stability.β-Adrenergic blockade combined with subcutaneous B-type natriuretic peptide: a promising approach to reduce ventricular arrhythmia in heart failure?Simultaneous activation of p38 MAPK and p42/44 MAPK by ATP stimulates the K+ current ITREK in cardiomyocytesTargeted deletion of Kv4.2 eliminates I(to,f) and results in electrical and molecular remodeling, with no evidence of ventricular hypertrophy or myocardial dysfunctionACE Inhibitor Delapril Prevents Ca(2+)-Dependent Blunting of IK1 and Ventricular Arrhythmia in Ischemic Heart Disease
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description
researcher ORCID ID = 0000-0001-9124-0066
@en
wetenschapper
@nl
name
Franck Aimond
@ast
Franck Aimond
@en
Franck Aimond
@es
Franck Aimond
@nl
type
label
Franck Aimond
@ast
Franck Aimond
@en
Franck Aimond
@es
Franck Aimond
@nl
prefLabel
Franck Aimond
@ast
Franck Aimond
@en
Franck Aimond
@es
Franck Aimond
@nl
P106
P31
P496
0000-0001-9124-0066