about
PPARγ as a therapeutic target to rescue mitochondrial function in neurological diseaseGene Transfer of Brain-derived Neurotrophic Factor (BDNF) Prevents Neurodegeneration Triggered by FXN DeficiencyGlutamate excitotoxicity and therapeutic targets for amyotrophic lateral sclerosis.PPARγ and PGC-1α as therapeutic targets in Parkinson's.Impaired mitochondrial homeostasis and neurodegeneration: towards new therapeutic targets?Infectious delivery and long-term persistence of transgene expression in the brain by a 135-kb iBAC-FXN genomic DNA expression vector.PPARγ activation rescues mitochondrial function from inhibition of complex I and loss of PINK1.AMPA receptor activation, but not the accumulation of endogenous extracellular glutamate, induces paralysis and motor neuron death in rat spinal cord in vivo.Calpain inhibition protects spinal motoneurons from the excitotoxic effects of AMPA in vivo.Paraptosis in human glioblastoma cell line induced by curcumin.Ca2+-permeable AMPA receptors and intracellular Ca2+ determine motoneuron vulnerability in rat spinal cord in vivoAntioxidants as a Potential Target against Inflammation and Oxidative Stress in Attention-Deficit/Hyperactivity DisorderAtomoxetine produces oxidative stress and alters mitochondrial function in human neuron-like cellsDifferences in substance use, psychiatric disorders and social factors between Mexican adolescents and young adults
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description
researcher ORCID ID = 0000-0003-4907-437X
@en
wetenschapper
@nl
name
Juan Carlos Corona
@ast
Juan Carlos Corona
@en
Juan Carlos Corona
@es
Juan Carlos Corona
@nl
type
label
Juan Carlos Corona
@ast
Juan Carlos Corona
@en
Juan Carlos Corona
@es
Juan Carlos Corona
@nl
prefLabel
Juan Carlos Corona
@ast
Juan Carlos Corona
@en
Juan Carlos Corona
@es
Juan Carlos Corona
@nl
P106
P1153
8116623700
P21
P31
P496
0000-0003-4907-437X