about
Deaf1 isoforms control the expression of genes encoding peripheral tissue antigens in the pancreatic lymph nodes during type 1 diabetesConcise Review: Cell-Based Therapies and Other Non-Traditional Approaches for Type 1 DiabetesToward beta cell replacement for diabetesA short pulse of IL-4 delivered by DCs electroporated with modified mRNA can both prevent and treat autoimmune diabetes in NOD mice.IFN-gamma gene expression is controlled by the architectural transcription factor HMGA1.Inflammation and hyperglycemia mediate Deaf1 splicing in the pancreatic lymph nodes via distinct pathways during type 1 diabetes.Reduced DEAF1 function during type 1 diabetes inhibits translation in lymph node stromal cells by suppressing Eif4g3.Lymphoid-tissue-specific homing of bone-marrow-derived dendritic cellsIt's time to bring dendritic cell therapy to type 1 diabetes.Efficient Presentation of Multiple Endogenous Epitopes to Both CD4+ and CD8+ Diabetogenic T Cells for ToleranceMultimodality imaging of T-cell hybridoma trafficking in collagen-induced arthritic mice: image-based estimation of the number of cells accumulating in mouse paws.[Superkines: cytokines displaying better targeted functions].TSG-6 protein expression in the pancreatic islets of NOD mice.Instruction of naive CD4+ T cells by polarized CD4+ T cells within dendritic cell clusters.A multi-epitope DNA vaccine enables a broad engagement of diabetogenic T cells for tolerance in Type 1 diabetesAltered Function of Antigen-Presenting Cells in Type 1 Diabetes: A Challenge for Antigen-Specific Immunotherapy?Humanized Mice Reveal New Insights Into the Thymic Selection of Human Autoreactive CD8 T CellsPPARγ deacetylation dissociates thiazolidinedione's metabolic benefits from its adverse effects.Local cooperation dominates over competition between CD4+ T cells of different antigen/MHC specificityThe immunological synapseMurine CD4+CD25+ regulatory T cells fail to undergo chromatin remodeling across the proximal promoter region of the IL-2 geneNF-κB in DCs: it takes effort to be immatureInitiating type I diabetes: new suspects in the lineup
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description
researcher ORCID ID = 0000-0002-8328-8155
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wetenschapper
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name
Remi Creusot
@ast
Remi Creusot
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Remi Creusot
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Remi Creusot
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type
label
Remi Creusot
@ast
Remi Creusot
@en
Remi Creusot
@es
Remi Creusot
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prefLabel
Remi Creusot
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Remi Creusot
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Remi Creusot
@es
Remi Creusot
@nl
P106
P31
P496
0000-0002-8328-8155