about
Modified mRNA directs the fate of heart progenitor cells and induces vascular regeneration after myocardial infarctionManipulation of a VEGF-Notch signaling circuit drives formation of functional vascular endothelial progenitors from human pluripotent stem cells.Reprogramming for cardiac regeneration.Synthetic chemically modified mRNA (modRNA): toward a new technology platform for cardiovascular biology and medicine.Highly efficient derivation of ventricular cardiomyocytes from induced pluripotent stem cells with a distinct epigenetic signature.A Src inhibitor regulates the cell cycle of human pluripotent stem cells and improves directed differentiation.Bioengineering heart muscle: a paradigm for regenerative medicineGenetic lineage tracing identifies in situ Kit-expressing cardiomyocytes.Infectious tolerance via the consumption of essential amino acids and mTOR signaling.Embryonic stem cells: overcoming the immunological barriers to cell replacement therapy.Mfsd2a+ hepatocytes repopulate the liver during injury and regenerationConcise review: Immune recognition of induced pluripotent stem cells.Protein kinases and associated pathways in pluripotent state and lineage differentiation.VEGF-A: the inductive angiogenic factor for development, regeneration and function of pancreatic beta cells.Cardiovascular regenerative therapeutics via synthetic paracrine factor modified mRNA.Integrative single-cell and cell-free plasma RNA transcriptomics elucidates placental cellular dynamicsPreexisting endothelial cells mediate cardiac neovascularization after injury.Deciphering the role of microRNAs in regulation of immune surveillance, self-tolerance and allograft transplantation.Tolerance induction to human stem cell transplants with extension to their differentiated progeny.Driving vascular endothelial cell fate of human multipotent Isl1+ heart progenitors with VEGF modified mRNA.A role for regulatory T cells in acceptance of ESC-derived tissues transplanted across an major histocompatibility complex barrier.GATA4 regulates Fgf16 to promote heart repair after injury.Individual Variation in Conditional β Cell Ablation Mice Contributes Significant Biases in Evaluating β Cell Functional Recovery.Dickkopf-3, a tissue-derived modulator of local T-cell responses.A Self-restricted CRISPR System to Reduce Off-target Effects.Genetic Modification of Human Pancreatic Progenitor Cells Through Modified mRNA.Replenishing the damaged heart with oxygen by nature-inspired photosynthesis.Fibroblasts in an endocardial fibroelastosis disease model mainly originate from mesenchymal derivatives of epicardium.Vascular Development and Regeneration in the Mammalian Heart.Endocardium Contributes to Cardiac Fat.Genetic Fate Mapping Defines the Vascular Potential of Endocardial Cells in the Adult Heart.Endocardial Cell Plasticity in Cardiac Development, Diseases and Regeneration.Genetic Lineage Tracing of Non-Myocyte Population by Dual Recombinases.DNase1 Does Not Appear to Play a Major Role in the Fragmentation of Plasma DNA in a Knockout Mouse Model.Regulatory T-Cells: Potential Regulator of Tissue Repair and Regeneration.Genetic lineage tracing of resident stem cells by DeaLTRegulatory T Cells Promote Apelin-Mediated Sprouting Angiogenesis in Type 2 DiabetesSingle-cell transcriptomics reveal that PD-1 mediates immune tolerance by regulating proliferation of regulatory T cellsLineage Tracing Reveals the Bipotency of SOX9 Hepatocytes during Liver RegenerationDeconstructive somatic cell nuclear transfer reveals novel regulatory T-cell subsets.
P50
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P50
description
onderzoeker
@nl
researcher ORCID ID = 0000-0002-1616-3643
@en
name
Kathy O Lui
@ast
Kathy O Lui
@en
Kathy O Lui
@nl
type
label
Kathy O Lui
@ast
Kathy O Lui
@en
Kathy O Lui
@nl
prefLabel
Kathy O Lui
@ast
Kathy O Lui
@en
Kathy O Lui
@nl
P106
P31
P496
0000-0002-1616-3643