about
A single-residue change in the HIV-1 V3 loop associated with maraviroc resistance impairs CCR5 binding affinity while increasing replicative capacity.Targeting spare CC chemokine receptor 5 (CCR5) as a principle to inhibit HIV-1 entry.HIV-1 exploits CCR5 conformational heterogeneity to escape inhibition by chemokinesBuffering deleterious polymorphisms in highly constrained parts of HIV-1 envelope by flexible regions.Guidelines for cloning, expression, purification and functional characterization of primary HIV-1 envelope glycoproteins.CCR5 structural plasticity shapes HIV-1 phenotypic propertiesSOS and IP Modifications Predominantly Affect the Yield but Not Other Properties of SOSIP.664 HIV-1 Env Glycoprotein Trimers
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description
researcher ORCID ID = 0000-0002-5552-8908
@en
name
Philippe Colin
@ast
Philippe Colin
@en
Philippe Colin
@es
Philippe Colin
@nl
type
label
Philippe Colin
@ast
Philippe Colin
@en
Philippe Colin
@es
Philippe Colin
@nl
prefLabel
Philippe Colin
@ast
Philippe Colin
@en
Philippe Colin
@es
Philippe Colin
@nl
P31
P496
0000-0002-5552-8908