about
Analgesia induced by the epigenetic drug, L-acetylcarnitine, outlasts the end of treatment in mouse models of chronic inflammatory and neuropathic pain.N-acetyl-cysteine, a drug that enhances the endogenous activation of group-II metabotropic glutamate receptors, inhibits nociceptive transmission in humans.Optical control of pain in vivo with a photoactive mGlu5 receptor negative allosteric modulatorType-1, but Not Type-5, Metabotropic Glutamate Receptors are Coupled to Polyphosphoinositide Hydrolysis in the Retina.N-Acetyl-cysteine causes analgesia by reinforcing the endogenous activation of type-2 metabotropic glutamate receptors.Pharmacological enhancement of mGlu1 metabotropic glutamate receptors causes a prolonged symptomatic benefit in a mouse model of spinocerebellar ataxia type 1.Metabotropic glutamate receptor-4 modulates adaptive immunity and restrains neuroinflammation.Cinnabarinic acid, an endogenous agonist of type-4 metabotropic glutamate receptor, suppresses experimental autoimmune encephalomyelitis in mice.Switch in the expression of mGlu1 and mGlu5 metabotropic glutamate receptors in the cerebellum of mice developing experimental autoimmune encephalomyelitis and in autoptic cerebellar samples from patients with multiple sclerosis.Correction: Optical control of pain in vivo with a photoactive mGlu5 receptor negative allosteric modulator.Expression of the K+/Cl- cotransporter, KCC2, in cerebellar Purkinje cells is regulated by group-I metabotropic glutamate receptors.
P50
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P50
description
Forscher
@de
chercheur
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investigador
@es
researcher
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հետազոտող
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研究者
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name
Serena Notartomaso
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Serena Notartomaso
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Serena Notartomaso
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Serena Notartomaso
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type
label
Serena Notartomaso
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Serena Notartomaso
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Serena Notartomaso
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Serena Notartomaso
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prefLabel
Serena Notartomaso
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Serena Notartomaso
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Serena Notartomaso
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Serena Notartomaso
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P496
0000-0003-4374-9233