about
The BCR-ABL oncoprotein potentially interacts with the xeroderma pigmentosum group B proteinCardiac fibroblasts are essential for the adaptive response of the murine heart to pressure overload.Three novel BMPR2 mutations associated with advanced pulmonary arterial hypertensionLgr5 Identifies Progenitor Cells Capable of Taste Bud Regeneration after Injury.Quantitative Measurement of GPCR Endocytosis via Pulse-Chase Covalent Labeling.Monitoring β-arrestin recruitment via β-lactamase enzyme fragment complementation: purification of peptide E as a low-affinity ligand for mammalian bombesin receptors.Dysbiosis and compositional alterations with aging in the gut microbiota of patients with heart failure.Hopx distinguishes hippocampal from lateral ventricle neural stem cells.Cellular Interplay between Cardiomyocytes and Nonmyocytes in Cardiac Remodeling.Pathophysiology and Management of Cardiovascular Manifestations in Marfan and Loeys-Dietz Syndromes.Peripartum type B aortic dissection in patients with Marfan syndrome who underwent aortic root replacement: a case series study.Plasticity of Hopx(+) type I alveolar cells to regenerate type II cells in the lung.Kruppel-like factor 5 causes cartilage degradation through transactivation of matrix metalloproteinase 9.Normalization of left ventricular parameters following combined pimobendan and carvedilol treatment in a case of unclassified cardiomyopathy with longstanding refractory status.Accelerated cardiac hypertrophy and renal damage induced by angiotensin II in adrenomedullin knockout mice.Protective effects of endogenous adrenomedullin on cardiac hypertrophy, fibrosis, and renal damage.Subarachnoid hemorrhage from mycotic aneurysms.[Atrial natriuretic peptide for therapy of heart failure].Takayasu myocarditis mediated by cytotoxic T lymphocytes.Periodontitis May Deteriorate Sinus of Valsalva Dilatation in Marfan Syndrome Patients.Evaluating Japanese patients with the Marfan syndrome using high-throughput microarray-based mutational analysis of fibrillin-1 gene.Distinct variants affecting differential splicing of TGFBR1 exon 5 cause either Loeys-Dietz syndrome or multiple self-healing squamous epithelioma.Response by Takeda et al to Letter Regarding Article, "Impact of Pathogenic FBN1 Variant Types on the Progression of Aortic Disease in Patients With Marfan Syndrome"Discovery of a Small Molecule to Increase Cardiomyocytes and Protect the Heart After Ischemic InjuryMarkedly increased levels of IL-6 and CA125 in pleural fluid of an elderly person with overlap syndrome of systemic sclerosis and systemic lupus erythematosusChronic advanced pulmonary hypertensionGamma-delta T-cell-mediated dilated cardiomyopathyAcyclic retinoid inhibits neointima formation through retinoic acid receptor beta-induced apoptosis[Efficacy and adverse reactions of atrial natriuretic peptide on kidney and cardiovascular system]Pulmonary thromboembolism in a patient with an anomalous right coronary artery as a cause of unusual biventricular myocardial infarctionDeterioration of atherosclerosis in mice lacking angiotensin II type 1A receptor in bone marrow-derived cellsBone marrow-derived cells contribute to vascular inflammation but do not differentiate into smooth muscle cell lineagesA case of localized IgG4-related thoracic periarteritis and recurrent nerve palsyIdiopathic retroperitoneal fibrosis, inflammatory aortic aneurysm, and inflammatory pericarditis--retrospective analysis of 11 case historiesCongenital contractural arachnodactyly complicated with aortic dilatation and dissection: Case report and review of literatureCongenital Contractural Arachnodactyly without FBN1 or FBN2 Gene Mutations Complicated by Dilated CardiomyopathyPathophysiology and Japanese clinical characteristics in Marfan syndromeAuthors' reply re: Peripartum type B aortic dissection in patients with Marfan syndrome who underwent aortic root replacement: a case series studyImpact of Pathogenic FBN1 Variant Types on the Progression of Aortic Disease in Patients With Marfan SyndromeProstaglandin E2 Signaling as a Target of Anti-Cardiac Fibrosis in Heart Failure Treatment
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P50
description
researcher ORCID ID = 0000-0003-4818-3347
@en
wetenschapper
@nl
name
Norifumi Takeda
@ast
Norifumi Takeda
@en
Norifumi Takeda
@es
Norifumi Takeda
@nl
type
label
Norifumi Takeda
@ast
Norifumi Takeda
@en
Norifumi Takeda
@es
Norifumi Takeda
@nl
prefLabel
Norifumi Takeda
@ast
Norifumi Takeda
@en
Norifumi Takeda
@es
Norifumi Takeda
@nl
P31
P496
0000-0003-4818-3347