about
The CACCC-binding protein KLF3/BKLF represses a subset of KLF1/EKLF target genes and is required for proper erythroid maturation in vivoRepression of chimeric transcripts emanating from endogenous retrotransposons by a sequence-specific transcription factor.Direct competition between DNA binding factors highlights the role of Krüppel-like Factor 1 in the erythroid/megakaryocyte switchImpaired B cell development in the absence of Krüppel-like factor 3.Generation of mice deficient in both KLF3/BKLF and KLF8 reveals a genetic interaction and a role for these factors in embryonic globin gene silencingEditing the genome to introduce a beneficial naturally occurring mutation associated with increased fetal globin.Transcription factors LRF and BCL11A independently repress expression of fetal hemoglobin.Krüppel-like Factor 3 (KLF3/BKLF) Is Required for Widespread Repression of the Inflammatory Modulator Galectin-3 (Lgals3).Cellular Reprogramming toward the Erythroid Lineage.Erratum to: Repression of chimeric transcripts emanating from endogenous retrotransposons by a sequence-specific transcription factor.
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description
Forscher
@de
chercheur
@fr
hulumtuese
@sq
investigador
@es
researcher
@en
wetenschapper
@nl
հետազոտող
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研究者
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name
Laura J Norton
@ast
Laura J Norton
@en
Laura J Norton
@es
Laura J Norton
@nl
type
label
Laura J Norton
@ast
Laura J Norton
@en
Laura J Norton
@es
Laura J Norton
@nl
prefLabel
Laura J Norton
@ast
Laura J Norton
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Laura J Norton
@es
Laura J Norton
@nl
P108
P1153
52364508800
P21
P31
P496
0000-0002-7950-381X