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5-Lipoxygenase negatively regulates Th1 response during Brucella abortus infection in miceSchistosoma mansoni Tegument (Smteg) Induces IL-10 and Modulates Experimental Airway Inflammation.Lack of IL-1 Receptor-Associated Kinase-4 Leads to Defective Th1 Cell Responses and Renders Mice Susceptible to Mycobacterial Infection.B. abortus RNA is the component involved in the down-modulation of MHC-I expression on human monocytes via TLR8 and the EGFR pathway.Nucleotide-binding oligomerization domain-2 (NOD2) regulates type-1 cytokine responses to Mycobacterium avium but is not required for host control of infection.Immunoproteasome subunits are required for CD8+ T cell function and host resistance to Brucella abortus infection in mice.Contribution of intercellular adhesion molecule 1 (ICAM-1) to control Mycobacterium avium infection.The use of gold nanorods as a new vaccine platform against schistosomiasis.Toll-like receptor 6 senses Mycobacterium avium and is required for efficient control of mycobacterial infection.The cGAS/STING Pathway Is Important for Dendritic Cell Activation but Is Not Essential to Induce Protective Immunity against Mycobacterium tuberculosis Infection.GM-CSF targeted immunomodulation affects host response to M. tuberculosis infection.Peptides containing T cell epitopes, derived from Sm14, but not from paramyosin, induce a Th1 type of immune response, reduction in liver pathology and partial protection against Schistosoma mansoni infection in miceSchistosoma mansoni antigens modulate the allergic response in a murine model of ovalbumin-induced airway inflammationAn intranasal administration of Lactococcus lactis strains expressing recombinant interleukin-10 modulates acute allergic airway inflammation in a murine modelBacterial RNA Contributes to the Down-Modulation of MHC-II Expression on Monocytes/Macrophages Diminishing CD4+ T Cell ResponsesAdvances in Immunology of Neglected Tropical Diseases: New Control Tools and Prospects for Disease EliminationBrucella abortus nitric oxide metabolite regulates inflammasome activation and IL-1β secretion in murine macrophagesJVA, an isoniazid analogue, is a bioactive compound against a clinical isolate of the Mycobacterium avium complexMycobacterium abscessus subsp. massiliense expressing bacterioferritin have improved resistance to stressful conditions
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description
researcher ORCID ID = 0000-0001-6885-3438
@en
wetenschapper
@nl
name
Fabio V Marinho
@ast
Fabio V Marinho
@en
Fabio V Marinho
@nl
type
label
Fabio V Marinho
@ast
Fabio V Marinho
@en
Fabio V Marinho
@nl
prefLabel
Fabio V Marinho
@ast
Fabio V Marinho
@en
Fabio V Marinho
@nl
P21
P31
P496
0000-0001-6885-3438