about
Alteration of growth factors and neuronal death in diabetic retinopathy: what we have learned so far.A lipidomic screen of hyperglycemia-treated HRECs links 12/15-Lipoxygenase to microvascular dysfunction during diabetic retinopathy via NADPH oxidase.SOD1 overexpression prevents acute hyperglycemia-induced cerebral myogenic dysfunction: relevance to contralateral hemisphere and stroke outcomes.The Chemokine Platelet Factor-4 Variant (PF-4var)/CXCL4L1 Inhibits Diabetes-Induced Blood-Retinal Barrier BreakdownDeletion of thioredoxin interacting protein (TXNIP) augments hyperoxia-induced vaso-obliteration in a mouse model of oxygen induced-retinopathy.Metformin treatment in the period after stroke prevents nitrative stress and restores angiogenic signaling in the brain in diabetes.Linagliptin treatment improves cerebrovascular function and remodeling and restores reduced cerebral perfusion in Type 2 diabetes.Matrix Metalloprotease 3 Exacerbates Hemorrhagic Transformation and Worsens Functional Outcomes in Hyperglycemic StrokePronerve growth factor induces angiogenesis via activation of TrkA: possible role in proliferative diabetic retinopathy.Cerebral neovascularization in diabetes: implications for stroke recovery and beyond.Linagliptin attenuates diabetes-induced cerebral pathological neovascularization in a blood glucose-independent manner: Potential role of ET-1.Cerebrovasculoprotective effects of azilsartan medoxomil in diabetes.Late dual endothelin receptor blockade with bosentan restores impaired cerebrovascular function in diabetes.Nox4 contributes to the hypoxia-mediated regulation of actin cytoskeleton in cerebrovascular smooth muscleCandesartan augments ischemia-induced proangiogenic state and results in sustained improvement after stroke.Dual endothelin receptor antagonism with bosentan reverses established vascular remodeling and dysfunctional angiogenesis in diabetic rats: relevance to glycemic control.Thioredoxin-interacting protein expression is required for VEGF-mediated angiogenic signal in endothelial cells.Peroxynitrite-Induced Tyrosine Nitration Contributes to Matrix Metalloprotease-3 Activation: Relevance to Hyperglycemic Ischemic Brain Injury and Tissue Plasminogen Activator.Enhanced VEGF signalling mediates cerebral neovascularisation via downregulation of guidance protein ROBO4 in a rat model of diabetes.Impact of Metabolic Diseases on Cerebral Circulation: Structural and Functional Consequences.Inhibition of Ephrin-B2 in brain pericytes decreases cerebral pathological neovascularization in diabetic ratsInhibition of Toll-Like Receptor-4 (TLR-4) Improves Neurobehavioral Outcomes After Acute Ischemic Stroke in Diabetic Rats: Possible Role of Vascular Endothelial TLR-4
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description
researcher ORCID ID = 0000-0001-9846-6623
@en
wetenschapper
@nl
name
Mohammed Abdelsaid
@ast
Mohammed Abdelsaid
@en
Mohammed Abdelsaid
@es
Mohammed Abdelsaid
@nl
type
label
Mohammed Abdelsaid
@ast
Mohammed Abdelsaid
@en
Mohammed Abdelsaid
@es
Mohammed Abdelsaid
@nl
prefLabel
Mohammed Abdelsaid
@ast
Mohammed Abdelsaid
@en
Mohammed Abdelsaid
@es
Mohammed Abdelsaid
@nl
P31
P496
0000-0001-9846-6623