about
Activation of PKCα and PI3K Kinases in Hypertrophic and Nodular Port Wine Stain LesionsDysregulation of mCD46 and sCD46 contribute to the pathogenesis of bullous pemphigoid.Topical rapamycin systematically suppresses the early stages of pulsed dye laser-induced angiogenesis pathways.Topical axitinib suppresses angiogenesis pathways induced by pulsed dye laser.The somatic GNAQ mutation (R183Q) is primarily located within the blood vessels of port wine stains.Dysregulated autophagy increased melanocyte sensitivity to H2O2-induced oxidative stress in vitiligo.Activation of Langerhans cells promotes the inflammation in imiquimod-induced psoriasis-like dermatitis.Clinic characteristics of psoriasis in China: a nationwide survey in over 12000 patients.TGFβ/SMAD/microRNA-486-3p Signaling Axis Mediates Keratin 17 Expression and Keratinocyte Hyperproliferation in Psoriasis.Oxidative stress drives CD8+ T-cell skin trafficking in patients with vitiligo through CXCL16 upregulation by activating the unfolded protein response in keratinocytes.Nrf2 promotes keratinocyte proliferation in psoriasis through up-regulation of Keratin 6, Keratin 16 and Keratin 17.Plexin-B1 and semaphorin 4D cooperate to promote cutaneous squamous cell carcinoma cell proliferation, migration and invasion.miR-196a-2 rs11614913 polymorphism is associated with vitiligo by affecting heterodimeric molecular complexes of Tyr and Tyrp1.Calcipotriol plus betamethasone dipropionate gel compared with calcipotriol scalp solution in the treatment of scalp psoriasis: a randomized, controlled trial investigating efficacy and safety in a Chinese population.Epidermal Dysfunction Leads to an Age-Associated Increase in Levels of Serum Inflammatory Cytokines.Oxidative Stress-Induced Chemokine Production Mediates CD8(+) T Cell Skin Trafficking in Vitiligo.Multiple pro-tumorigenic functions of the human minor Histocompatibility Antigen-1 (HA-1) in melanoma progression.Vascular endothelial growth factor driving aberrant keratin expression pattern contributes to the pathogenesis of psoriasis.CD100-Plexin-B2 Promotes the Inflammation in Psoriasis by Activating NF-κB and the Inflammasome in Keratinocytes.Semaphorin4D Drives CD8+ T-Cell Lesional Trafficking in Oral Lichen Planus via CXCL9/CXCL10 Upregulations in Oral Keratinocytes.NB-UVB irradiation downregulates keratin-17 expression in keratinocytes by inhibiting the ERK1/2 and STAT3 signaling pathways.Dysfunction of CD19+CD24hiCD27+ B regulatory cells in patients with bullous pemphigoid.Ubiquitination-proteasome system: A new player in the pathogenesis of psoriasis and clinical implications.Semaphorin 4D from CD15+ Granulocytes via ADAM10-Induced Cleavage Contributes to Antibody Production in Bullous Pemphigoid.A Novel COL7A1 Mutation in a Chinese Family with Epidermolysis Bullosa Pruriginosa.Sema4D is required in both the adaptive and innate immune responses of contact hypersensitivity.Down-regulated TRPV1 expression contributes to melanoma growth via calcineurin-ATF3-p53 pathway.Increased expression of NLRP3 inflammasome components and interleukin-18 in patients with bullous pemphigoid.MicroRNA-17-92 cluster promotes the proliferation and the chemokine production of keratinocytes: implication for the pathogenesis of psoriasis.Proinflammatory role of blister fluid-derived exosomes in bullous pemphigoid.HO-1 regulates the function of Treg: Association with the immune intolerance in vitiligoPsoriatic mesenchymal stem cells demonstrate an enhanced ability to differentiate into vascular endothelial cellsKeratin 17 in disease pathogenesis: from cancer to dermatosesNeutrophil Extracellular Traps Promote Inflammatory Responses in Psoriasis via Activating Epidermal TLR4/IL-36R CrosstalkIdentification of Immunodominant Th2-Cell Epitopes in Chinese Patients with Bullous PemphigoidE3 Ligase Trim21 Ubiquitylates and Stabilizes Keratin 17 to Induce STAT3 Activation in PsoriasisActivated NLR family pyrin domain containing 3 (NLRP3) inflammasome in keratinocytes promotes cutaneous T-cell response in patients with vitiligoNeutrophil exosomes enhance the skin autoinflammation in generalized pustular psoriasis via activating keratinocytesTopical applications of an emollient reduce circulating pro-inflammatory cytokine levels in chronically aged humans: a pilot clinical studyA combination of 2940-nm laser and photodynamic therapy for treatment of recalcitrant facial flat warts
P50
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P50
description
researcher ORCID ID = 0000-0002-5842-8080
@en
name
Gang Wang
@ast
Gang Wang
@en
Gang Wang
@es
Gang Wang
@nl
type
label
Gang Wang
@ast
Gang Wang
@en
Gang Wang
@es
Gang Wang
@nl
prefLabel
Gang Wang
@ast
Gang Wang
@en
Gang Wang
@es
Gang Wang
@nl
P1153
57197760259
P31
P496
0000-0002-5842-8080