about
Relevance of the Carotid Body Chemoreflex in the Progression of Heart FailureRole of the carotid body in the pathophysiology of heart failureChronic intermittent hypoxia augments chemoreflex control of sympathetic activity: role of the angiotensin II type 1 receptor.Time-dependent adaptation in the hemodynamic response to hypoxia.Time course of intermittent hypoxia-induced impairments in resistance artery structure and function.Central role of carotid body chemoreceptors in disordered breathing and cardiorenal dysfunction in chronic heart failure.Exercise training attenuates chemoreflex-mediated reductions of renal blood flow in heart failure.Mechanisms of carotid body chemoreflex dysfunction during heart failure.Exercise training normalizes renal blood flow responses to acute hypoxia in experimental heart failure: role of the α1-adrenergic receptorInhibition of hydrogen sulfide restores normal breathing stability and improves autonomic control during experimental heart failure.Carotid chemoreceptor ablation improves survival in heart failure: rescuing autonomic control of cardiorespiratory function.Simvastatin treatment attenuates increased respiratory variability and apnea/hypopnea index in rats with chronic heart failure.Selective carotid body ablation in experimental heart failure: a new therapeutic tool to improve cardiorespiratory control.Role of the Carotid Body Chemoreflex in the Pathophysiology of Heart Failure: A Perspective from Animal Studies.Contribution of peripheral and central chemoreceptors to sympatho-excitation in heart failure.Cardiac diastolic and autonomic dysfunction are aggravated by central chemoreflex activation in heart failure with preserved ejection fraction rats.Carotid Body-Mediated Chemoreflex Drive in The Setting of low and High Output Heart Failure.KLF2 mediates enhanced chemoreflex sensitivity, disordered breathing and autonomic dysregulation in heart failure.Fat feeding facilitates hot bodies, but is resistance futile?Revisiting the Physiological Effects of Exercise Training on Autonomic Regulation and Chemoreflex Control in Heart Failure: Does Ejection Fraction Matter?Reply from Noah J. Marcus, Rodrigo Del Rio and Harold D. Schultz.Exercise training improves cardiac autonomic control, cardiac function, and arrhythmogenesis in rats with preserved-ejection fraction heart failure.Carotid body denervation improves autonomic and cardiac function and attenuates disordered breathing in congestive heart failure.Topical Application of Connexin43 Hemichannel Blocker Reduces Carotid Body-Mediated Chemoreflex Drive in RatsVentilatory and Autonomic Regulation in Sleep Apnea Syndrome: A Potential Protective Role for Erythropoietin?Aberrant reflex mechanisms contributing to reno-vascular hypertension: a pain in the neck?Episodic stimulation of central chemoreceptor neurons elicits disordered breathing and autonomic dysfunction in volume overload heart failureRostral ventrolateral medullary catecholaminergic neurones mediate irregular breathing pattern in volume overload heart failure ratsAblation of brainstem C1 neurons improves cardiac function in volume overload heart failureNeuroinflammation in heart failure: new insights for an old diseaseHeart rate variability alterations in infants with spontaneous hypertonia
P50
Q26771307-3F64D5B1-CE40-4410-956E-E1B13DA9A2EAQ26853502-A7139361-86CE-4CD4-9040-7FA971EE9C89Q30475820-1AC0D11A-1F44-43A6-80B6-2F9EC0C0E9A1Q30484430-E0742DCA-8374-41A8-9D96-83FE4D909B5BQ33652144-8346BE40-D389-4F7E-9B99-DE7BF0ADB4EBQ34562968-5E34B389-AD40-4E07-B311-D9009D4916E5Q35859286-502C5BA9-D116-4B35-BFC7-C01970974456Q36263357-DA5EB997-8013-4376-AFAA-5914A30C8966Q36541471-455AF025-E83E-4716-85FD-22D17C2704D7Q36850784-51F73145-95B5-40D7-9999-998EAECD09A6Q37407921-6234C07D-4041-49E7-AB89-2CD1420808EAQ37712597-958D9749-0718-45AF-8187-36B6C194890DQ38268354-E7A3B8CB-718A-420E-9C0C-EF370D88C4A0Q38572976-AEE02BAA-A054-4E2D-9467-8AC17B09C62EQ38842860-CB32861C-A638-4ACF-A591-AF2CC70CF31FQ38970479-75902E68-A077-4B16-9D5F-E94C1268F61BQ41387881-A8BB1AC0-48B6-4608-B041-33DE524A2BEFQ41918181-C887FBCE-A860-4E13-BD4F-2C1A040408E2Q48144579-4F6328B0-B08A-4A14-A84F-AC9C0C9AAB43Q49789531-3F14FEF7-E9C0-4C47-B059-09FABD7646F0Q51729618-45C3057C-8213-4197-9C8F-E9870DD927BCQ52091312-D4E7B224-5DF2-4AB8-B129-00C90AD2CFA2Q54576764-97ABFB26-7C42-403C-9A11-08B5F2CB74A4Q57826225-E6A5DB20-D081-4FE0-A44D-F07691EA3394Q58121715-58426953-F051-4422-BE49-A202526ED4A9Q88664827-82C73F18-8411-48E6-BF41-08EF1C971637Q90741816-3525D1C3-48E1-478A-8388-64211359539DQ90899268-1507F609-0520-41B9-A81E-DC0D143063D8Q90970001-E558859E-8D4D-4209-8A96-79CFE68FA8EDQ91036715-28ED9046-435E-4A65-9FCC-4097088CB57DQ91520892-0C9C1298-7324-4F20-B7FB-A6F48C88E8C7
P50
description
researcher ORCID ID = 0000-0003-0362-0662
@en
name
Noah J Marcus
@ast
Noah J Marcus
@en
Noah J Marcus
@nl
type
label
Noah J Marcus
@ast
Noah J Marcus
@en
Noah J Marcus
@nl
prefLabel
Noah J Marcus
@ast
Noah J Marcus
@en
Noah J Marcus
@nl
P21
P31
P496
0000-0003-0362-0662