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Addressing Emerging Risks: Scientific and Regulatory Challenges Associated with Environmentally Persistent Free RadicalsCrawling with Virus: Translational Insights from a Neonatal Mouse Model on the Pathogenesis of Respiratory Syncytial Virus in InfantsA causative relationship exists between eosinophils and the development of allergic pulmonary pathologies in the mouseModel combustion-generated particulate matter containing persistent free radicals redox cycle to produce reactive oxygen speciesCombustion By-Products and their Health Effects--combustion engineering and global health in the 21st century: issues and challenges.Mouse eosinophil-associated ribonucleases: a unique subfamily expressed during hematopoiesisIn vitro and in vivo assessment of pulmonary risk associated with exposure to combustion generated fine particlesAcute Lung Injury Results from Innate Sensing of Viruses by an ER Stress PathwayLimited type I interferons and plasmacytoid dendritic cells during neonatal respiratory syncytial virus infection permit immunopathogenesis upon reinfection.Deficiency of the two-pore-domain potassium channel TREK-1 promotes hyperoxia-induced lung injury.Exposure to electronic cigarettes impairs pulmonary anti-bacterial and anti-viral defenses in a mouse model.Exposure to combustion generated environmentally persistent free radicals enhances severity of influenza virus infectionOrigin and health impacts of emissions of toxic by-products and fine particles from combustion and thermal treatment of hazardous wastes and materials.Environmentally persistent free radicals induce airway hyperresponsiveness in neonatal rat lungs.Impaired gamma delta T cell-derived IL-17A and inflammasome activation during early respiratory syncytial virus infection in infantsSerotonin 5-HT₂ receptor activation prevents allergic asthma in a mouse modelRespiratory Syncytial Virus Disease Is Mediated by Age-Variable IL-33.Exposure to Deepwater Horizon Crude Oil Burnoff Particulate Matter Induces Pulmonary Inflammation and Alters Adaptive Immune ResponseBuilding a better neonatal mouse model to understand infant respiratory syncytial virus disease.A Neonatal Murine Model of MRSA Pneumonia.Regulation of inflammatory biomarkers by intravenous methylprednisolone in pediatric ARDS patients: Results from a double-blind, placebo-controlled randomized pilot trial.Using an External Exposome Framework to Examine Pregnancy-Related Morbidities and Mortalities: Implications for Health Disparities Research.Plasma Biomarker Analysis in Pediatric ARDS: Generating Future Framework from a Pilot Randomized Control Trial of Methylprednisolone: A Framework for Identifying Plasma Biomarkers Related to Clinical Outcomes in Pediatric ARDS.IL-4Rα on CD4+ T cells plays a pathogenic role in respiratory syncytial virus reinfection in mice infected initially as neonatesMaternal exposure to combustion generated PM inhibits pulmonary Th1 maturation and concomitantly enhances postnatal asthma development in offspring.Exposure to environmentally persistent free radicals during gestation lowers energy expenditure and impairs skeletal muscle mitochondrial function in adult mice.Chronic alcohol induces M2 polarization enhancing pulmonary disease caused by exposure to particulate air pollutionAutophagy is essential for ultrafine particle-induced inflammation and mucus hyperproduction in airway epithelium.Regulatory T cells and IL10 suppress pulmonary host defense during early-life exposure to radical containing combustion derived ultrafine particulate matter.Hepatitis E antibodies in laboratory rabbits from 2 US vendors.A Scalable Field Study Protocol and Rationale for Passive Ambient Air Sampling: A Spatial Phytosampling for Leaf Data Collection.Prolonged viral replication and longitudinal viral dynamic differences among respiratory syncytial virus (RSV) infected infants.IL-4Rα on dendritic cells in neonates and Th2 immunopathology in respiratory syncytial virus infection.Early-life exposure to combustion-derived particulate matter causes pulmonary immunosuppression.Spatiotemporal patterns of childhood asthma hospitalization and utilization in Memphis Metropolitan Area from 2005 to 2015.Immunomodulation with IL-4R alpha antisense oligonucleotide prevents respiratory syncytial virus-mediated pulmonary disease.Environmentally persistent free radicals amplify ultrafine particle mediated cellular oxidative stress and cytotoxicity.Extensive eosinophil degranulation and peroxidase-mediated oxidation of airway proteins do not occur in a mouse ovalbumin-challenge model of pulmonary inflammation.Methacholine-induced airway hyperresponsiveness is dependent on Galphaq signaling.14th congress of combustion by-products and their health effects-origin, fate, and health effects of combustion-related air pollutants in the coming era of bio-based energy sources.
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description
onderzoeker
@nl
researcher
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հետազոտող
@hy
name
Stephania A Cormier
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Stephania A Cormier
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Stephania A Cormier
@es
Stephania A Cormier
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type
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Stephania A Cormier
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Stephania A Cormier
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Stephania A Cormier
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Stephania A Cormier
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prefLabel
Stephania A Cormier
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Stephania A Cormier
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Stephania A Cormier
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Stephania A Cormier
@nl
P106
P31
P496
0000-0002-6050-6172