about
EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency.Persistent KSHV Infection Increases EBV-Associated Tumor Formation In Vivo via Enhanced EBV Lytic Gene Expression.Epstein-Barr virus nuclear protein EBNA3C directly induces expression of AID and somatic mutations in B cells.The Cooperative Functions of the EBNA3 Proteins Are Central to EBV Persistence and Latency.
P50
description
hulumtuese
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Christine T Styles
@ast
Christine T Styles
@en
Christine T Styles
@es
Christine T Styles
@nl
type
label
Christine T Styles
@ast
Christine T Styles
@en
Christine T Styles
@es
Christine T Styles
@nl
prefLabel
Christine T Styles
@ast
Christine T Styles
@en
Christine T Styles
@es
Christine T Styles
@nl
P106
P21
P31
P496
0000-0001-7337-2540