about
A senescence-like cell-cycle arrest occurs during megakaryocytic maturation: implications for physiological and pathological megakaryocytic proliferationThe inactive X chromosome is epigenetically unstable and transcriptionally labile in breast cancerLigand-independent thrombopoietin mutant receptor requires cell surface localization for endogenous activity.X-chromosome inactivation in development and cancer.Induction of myeloproliferative disorder and myelofibrosis by thrombopoietin receptor W515 mutants is mediated by cytosolic tyrosine 112 of the receptor.Proteasome inhibitor bortezomib impairs both myelofibrosis and osteosclerosis induced by high thrombopoietin levels in mice.SOCS3 inhibits TPO-stimulated, but not spontaneous, megakaryocytic growth in primary myelofibrosis.Evidence for MPL W515L/K mutations in hematopoietic stem cells in primitive myelofibrosisNew mutations of MPL in primitive myelofibrosis: only the MPL W515 mutations promote a G1/S-phase transitionThe hematopoietic stem cell compartment of JAK2V617F-positive myeloproliferative disorders is a reflection of disease heterogeneity
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Ronan Chaligné
@ast
Ronan Chaligné
@en
Ronan Chaligné
@es
Ronan Chaligné
@nl
type
label
Ronan Chaligné
@ast
Ronan Chaligné
@en
Ronan Chaligné
@es
Ronan Chaligné
@nl
prefLabel
Ronan Chaligné
@ast
Ronan Chaligné
@en
Ronan Chaligné
@es
Ronan Chaligné
@nl
P106
P31
P496
0000-0001-5788-5848