about
mtRF1a is a human mitochondrial translation release factor decoding the major termination codons UAA and UAGJNK/SAPK Signaling Is Essential for Efficient Reprogramming of Human Fibroblasts to Induced Pluripotent Stem CellsLysosome-mediated processing of chromatin in senescence.SQSTM1/p62 mediates crosstalk between autophagy and the UPS in DNA repair.Cellular senescence drives age-dependent hepatic steatosis.Chronic inflammation induces telomere dysfunction and accelerates ageing in mice.Mitochondria are required for pro-ageing features of the senescent phenotype.A stochastic step model of replicative senescence explains ROS production rate in ageing cell populationsControl of TSC2-Rheb signaling axis by arginine regulates mTORC1 activity.CDK1 plays an important role in the maintenance of pluripotency and genomic stability in human pluripotent stem cellsDNA damage response at telomeres contributes to lung aging and chronic obstructive pulmonary disease.DNA damage in telomeres and mitochondria during cellular senescence: is there a connection?Methods for cell sorting of young and senescent cells.Cellular senescence mediates fibrotic pulmonary diseaseTelomeres, oxidative stress and inflammatory factors: partners in cellular senescence?Mitochondria: Are they causal players in cellular senescence?Depletion of mitochondria in mammalian cells through enforced mitophagy.Targeting the SASP to combat ageing: Mitochondria as possible intracellular allies?Telomeres and Cell Senescence - Size Matters Not.Measuring reactive oxygen species in senescent cells.Demystifying the role of mitochondria in senescence.Telomeres are favoured targets of a persistent DNA damage response in ageing and stress-induced senescence.Stress, cell senescence and organismal ageing.Powering senescence: The ugly side of mitochondria.Detecting senescence: a new method for an old pigment.Telomere Dysfunction and Senescence-associated Pathways in Bronchiectasis.Mechanisms driving the ageing heart.Mitochondrial dysfunction is a possible cause of accelerated senescence of mesothelial cells exposed to high glucose.Mitochondria, telomeres and cell senescence: Implications for lung ageing and disease.Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis.Portugal gets ready to embrace ageing research.Mitochondria and ageing: winning and losing in the numbers game.Mitochondria and cellular senescence: Implications for musculoskeletal ageingCell Sorting of Young and Senescent CellsTelomerase does not counteract telomere shortening but protects mitochondrial function under oxidative stressPremature senescence of mesothelial cells is associated with non-telomeric DNA damageObesity-Induced Cellular Senescence Drives Anxiety and Impairs NeurogenesisLength-independent telomere damage drives post-mitotic cardiomyocyte senescenceThe innate immune sensor Toll-like receptor 2 controls the senescence-associated secretory phenotypeMonoamine oxidase-A is a novel driver of stress-induced premature senescence through inhibition of parkin-mediated mitophagy
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description
researcher
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wetenschapper
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հետազոտող
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name
Joao F Passos
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Joao F Passos
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Joao F Passos
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Joao F Passos
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Joao F Passos
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Joao F Passos
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Joao F Passos
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Joao F Passos
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Joao F Passos
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Joao F Passos
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Joao F Passos
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Joao F Passos
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P106
P21
P31
P496
0000-0001-8765-1890