about
TFIIH transcription factor, a target for the Rift Valley hemorrhagic fever virus.A SAP30 complex inhibits IFN-beta expression in Rift Valley fever virus infected cells.TFIIH subunit alterations causing xeroderma pigmentosum and trichothiodystrophy specifically disturb several steps during transcription.Influenza virus NS1 protein binds cellular DNA to block transcription of antiviral genes.XPG and XPF endonucleases trigger chromatin looping and DNA demethylation for accurate expression of activated genes.NER factors are recruited to active promoters and facilitate chromatin modification for transcription in the absence of exogenous genotoxic attack.Transcription without XPB Establishes a Unified Helicase-Independent Mechanism of Promoter Opening in Eukaryotic Gene Expression.XPC is an RNA polymerase II cofactor recruiting ATAC to promoters by interacting with E2F1.Functional interplay between TFIIH and KAT2A regulates higher-order chromatin structure and class II gene expressionPoly (ADP-ribose) glycohydrolase regulates retinoic acid receptor-mediated gene expressionActivation of P-TEFb by RBM7: To Live or Let DieDNA repair complex licenses acetylation of H2A.Z.1 by KAT2A during transcription
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Nicolas Le May
@ast
Nicolas Le May
@en
Nicolas Le May
@es
Nicolas Le May
@nl
type
label
Nicolas Le May
@ast
Nicolas Le May
@en
Nicolas Le May
@es
Nicolas Le May
@nl
prefLabel
Nicolas Le May
@ast
Nicolas Le May
@en
Nicolas Le May
@es
Nicolas Le May
@nl
P106
P21
P31
P496
0000-0002-0172-8752