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Gene Therapy Corrects Mitochondrial Dysfunction in Hematopoietic Progenitor Cells and Fibroblasts from Coq9R239X Mice.CoQ deficiency causes disruption of mitochondrial sulfide oxidation, a new pathomechanism associated with this syndrome.Mitochondrial impairment and melatonin protection in parkinsonian mice do not depend of inducible or neuronal nitric oxide synthasesReduction in the levels of CoQ biosynthetic proteins is related to an increase in lifespan without evidence of hepatic mitohormesisβ-RA reduces DMQ/CoQ ratio and rescues the encephalopathic phenotype in miceRapamycin administration is not a valid therapeutic strategy for every case of mitochondrial diseaseThe Paradox of Coenzyme Q10 in Aging
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description
onderzoeker
@nl
researcher
@en
հետազոտող
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name
Eliana Barriocanal-Casado
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Eliana Barriocanal-Casado
@en
Eliana Barriocanal-Casado
@es
Eliana Barriocanal-Casado
@nl
type
label
Eliana Barriocanal-Casado
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Eliana Barriocanal-Casado
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Eliana Barriocanal-Casado
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Eliana Barriocanal-Casado
@nl
prefLabel
Eliana Barriocanal-Casado
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Eliana Barriocanal-Casado
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Eliana Barriocanal-Casado
@es
Eliana Barriocanal-Casado
@nl
P106
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57190018288
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P496
0000-0001-8828-1182