about
Isolation and identification of the human homolog of a new p53-binding protein, MdmxThe cdk5/p35 kinase is essential for neurite outgrowth during neuronal differentiationTissue-specific transcript annotation and expression profiling with complementary next-generation sequencing technologies.Genes involved in the osteoarthritis process identified through genome wide expression analysis in articular cartilage; the RAAK study.A gain of function mutation in TNFRSF11B encoding osteoprotegerin causes osteoarthritis with chondrocalcinosis.Aberrant Calreticulin Expression in Articular Cartilage of Dio2 Deficient Mice.Associations between joint effusion in the knee and gene expression levels in the circulation: a meta-analysis.Implementation of Functional Genomics for Bench-to-Bedside Transition in Osteoarthritis.Integrative epigenomics, transcriptomics and proteomics of patient chondrocytes reveal genes and pathways involved in osteoarthritis.Translating genomics into mechanisms of disease: Osteoarthritis.Neo-cartilage engineered from primary chondrocytes is epigenetically similar to autologous cartilage, in contrast to using mesenchymal stem cells.The role of epigenetics in osteoarthritis: current perspective.Knee and hip articular cartilage have distinct epigenomic landscapes: implications for future cartilage regeneration approaches.Transcriptional associations of osteoarthritis-mediated loss of epigenetic control in articular cartilage.The effect of forced exercise on knee joints in Dio2(-/-) mice: type II iodothyronine deiodinase-deficient mice are less prone to develop OA-like cartilage damage upon excessive mechanical stress.Inactivation of the p53 pathway in retinoblastoma.Underlying molecular mechanisms of DIO2 susceptibility in symptomatic osteoarthritis.Genes expressed in blood link osteoarthritis with apoptotic pathways.Genome-wide analyses identify a role for SLC17A4 and AADAT in thyroid hormone regulationComparative study of the p53-mdm2 and p53-MDMX interfacesIncreased type II deiodinase protein in OA-affected cartilage and allelic imbalance of OA risk polymorphism rs225014 at DIO2 in human OA joint tissues
P50
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P50
description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Yolande F. M. Ramos
@ast
Yolande F. M. Ramos
@en
Yolande F. M. Ramos
@es
Yolande F. M. Ramos
@nl
type
label
Yolande F. M. Ramos
@ast
Yolande F. M. Ramos
@en
Yolande F. M. Ramos
@es
Yolande F. M. Ramos
@nl
prefLabel
Yolande F. M. Ramos
@ast
Yolande F. M. Ramos
@en
Yolande F. M. Ramos
@es
Yolande F. M. Ramos
@nl
P1053
C-3774-2018
P106
P31
P496
0000-0003-1459-413X