about
Induction of micronuclei in V79 cells by fractions of roofing asphalt fume condensate.Detection of genomic instability in lung cancer tissues by random amplified polymorphic DNA analysisTransduction of skeletal muscles with common reporter genes can promote muscle fiber degeneration and inflammationmiR-206 represses hypertrophy of myogenic cells but not muscle fibers via inhibition of HDAC4Arresting angiotensin type 1 receptors.Development of novel activin-targeted therapeutics.When 6 is 9: 'uncoupled' AT1 receptors turn signalling on its head.Evaluation of follistatin as a therapeutic in models of skeletal muscle atrophy associated with denervation and tenotomy.Lentiviral transduction of rat Sertoli cells as a means to modify gene expression.Type 1 angiotensin receptor pharmacology: signaling beyond G proteins.Using AAV vectors expressing the β2-adrenoceptor or associated Gα proteins to modulate skeletal muscle mass and muscle fibre size.Integrated expression analysis of muscle hypertrophy identifies Asb2 as a negative regulator of muscle mass.The bone morphogenetic protein axis is a positive regulator of skeletal muscle mass.A functional siRNA screen identifies genes modulating angiotensin II-mediated EGFR transactivation.Elevated expression of activins promotes muscle wasting and cachexia.Specific targeting of TGF-β family ligands demonstrates distinct roles in the regulation of muscle mass in health and disease.Phosphoinositide 3-kinase (p110α) gene delivery limits diabetes-induced cardiac NADPH oxidase and cardiomyopathy in a mouse model with established diastolic dysfunction.Smad7 gene delivery prevents muscle wasting associated with cancer cachexia in mice.Differential Effects of IL6 and Activin A in the Development of Cancer-Associated Cachexia.Deficiency in Apoptosis-Inducing Factor Recapitulates Chronic Kidney Disease via Aberrant Mitochondrial Homeostasis.The atypical 'b' splice variant of phospholipase Cβ1 promotes cardiac contractile dysfunction.The Hippo pathway effector YAP is a critical regulator of skeletal muscle fibre size.Side-chain substitutions within angiotensin II reveal different requirements for signaling, internalization, and phosphorylation of type 1A angiotensin receptors.Gene delivery of medium chain acyl-coenzyme A dehydrogenase (MCAD) induces physiological cardiac hypertrophy and protects against pathological remodelling.Skeletal Muscle-Specific Overexpression of Heat Shock Protein 72 Improves Skeletal Muscle Insulin-Stimulated Glucose Uptake But Does Not Alter Whole Body Metabolism.Determination of the exact molecular requirements for type 1 angiotensin receptor epidermal growth factor receptor transactivation and cardiomyocyte hypertrophy.Dissociation of Pentameric to Monomeric C-Reactive Protein on Activated Platelets Localizes Inflammation to Atherosclerotic PlaquesHeteromerization of angiotensin receptors changes trafficking and arrestin recruitment profiles
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description
researcher
@en
name
Hongwei W. Qian
@en
Qian HW
@nl
type
label
Hongwei W. Qian
@en
Qian HW
@nl
altLabel
Qian HW
@en
prefLabel
Hongwei W. Qian
@en
Qian HW
@nl
P106
P31
P496
0000-0002-5991-6860