about
Activation of p53 in cervical carcinoma cells by small molecules.Inhibition of c-Abl kinase activity renders cancer cells highly sensitive to mitoxantroneCollagenase-3 (MMP-13) is expressed by tumor cells in invasive vulvar squamous cell carcinomas.Characterization of keratin and cell cycle protein expression in cell lines from squamous intraepithelial lesions progressing towards a malignant phenotype.Activation of p53 in cervical cancer cells by human papillomavirus E6 RNA interference is transient, but can be sustained by inhibiting endogenous nuclear export-dependent p53 antagonists.DNA topoisomerase I is a cofactor for c-Jun in the regulation of epidermal growth factor receptor expression and cancer cell proliferationMutations of TP53 do not correlate with the sensitivity to paclitaxel--a study using 27 gynaecological cancer cell lines.p53 mutations and presence of HPV DNA do not correlate with radiosensitivity of gynecological cancer cell lines.Mutation of tumor suppressor gene p53 is frequently found in vulvar carcinoma cells.Rapid and effective detection of mutations in the p53 gene using nonradioactive single-strand conformation polymorphism (SSCP) technique applied on PhastSystem.Novel action modality of the diterpenoid anisomelic acid causes depletion of E6 and E7 viral oncoproteins in HPV-transformed cervical carcinoma cells.Chemoradiation of cervical cancer cells: targeting human papillomavirus E6 and p53 leads to either augmented or attenuated apoptosis depending on the platinum carrier ligand.Response of estrogen receptor-positive intraabdominal fibromatosis to aromatase inhibitor therapy.Chemotherapy compounds in cervical cancer cells primed by reconstitution of p53 function after short interfering RNA-mediated degradation of human papillomavirus 18 E6 mRNA: opposite effect of siRNA in combination with different drugs.A Functional Homologous Recombination Assay Predicts Primary Chemotherapy Response and Long-Term Survival in Ovarian Cancer PatientsTime to go functional! Determining tumors' DNA repair capacityIdentifying differentially methylated sites in samples with varying tumor purityDecorin Expression in Human Vulva Carcinoma: Oncosuppressive Effect of Decorin cDNA Transduction on Carcinoma CellsBaltic Sea cyanobacterial bloom contains denitrification and nitrification genes, but has negligible denitrification activityType I diabetic pregnancy and subclinical human papillomavirus infectionExpression of cell-cycle proteins p53, p21 (WAF-1), PCNA and Ki-67 in benign, premalignant and malignant skin lesions with implicated HPV involvementHysterectomy for gynaecological cancer: a follow-up study of subjective and objective outcomeThe cytotoxicity of chemotherapy drugs varies in cervical cancer cells depending on the p53 status[Update on current care guidelines: ovarian cancer]Mathematical Modeling Predicts Response to Chemotherapy and Drug Combinations in Ovarian CancerAssociation of Vulvar Melanoma with Lichen SclerosusBehavior of FDG-avid supradiaphragmatic lymph nodes in PET/CT throughout primary therapy in advanced serous epithelial ovarian cancer: a prospective studyOpen Source Infrastructure for Health Care Data Integration and Machine Learning AnalysesNiraparib plus bevacizumab versus niraparib alone for platinum-sensitive recurrent ovarian cancer (NSGO-AVANOVA2/ENGOT-ov24): a randomised, phase 2, superiority trial
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P50
description
researcher
@en
name
S H Hietanen
@en
S H Hietanen
@nl
type
label
S H Hietanen
@en
S H Hietanen
@nl
prefLabel
S H Hietanen
@en
S H Hietanen
@nl
P106
P31
P496
0000-0003-4734-4743