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Human surfactant protein A2 gene mutations impair dimmer/trimer assembly leading to deficiency in protein sialylation and secretionChronic Normobaric Hypoxia Induces Pulmonary Hypertension in Rats: Role of NF-κB.Surfactant protein A2 mutations associated with pulmonary fibrosis lead to protein instability and endoplasmic reticulum stress.Engineering vascular tissue with functional smooth muscle cells derived from human iPS cells and nanofibrous scaffoldsCardiac β2-Adrenergic Receptor Phosphorylation at Ser355/356 Regulates Receptor Internalization and Functional Resensitization.Enhanced expression of TREM-1 in splenic cDCs in lupus prone mice and it was modulated by miRNA-150.Mutations of rat surfactant protein A have distinct effects on its glycosylation, secretion, aggregation and degradation.Interleukin-6 family of cytokines mediates isoproterenol-induced delayed STAT3 activation in mouse heart.Norepinephrine- and epinephrine-induced distinct beta2-adrenoceptor signaling is dictated by GRK2 phosphorylation in cardiomyocytes.Noncanonical cAMP pathway and p38 MAPK mediate beta2-adrenergic receptor-induced IL-6 production in neonatal mouse cardiac fibroblasts.Amelioration of apelin-13 in chronic normobaric hypoxia-induced anxiety-like behavior is associated with an inhibition of NF-κB in the hippocampus.SIRT1 Mediates Apelin-13 in Ameliorating Chronic Normobaric Hypoxia-induced Anxiety-like Behavior by Suppressing NF-κB Pathway in Mice Hippocampus.Chemical chaperone 4-phenylbutyric acid alleviates the aggregation of human familial pulmonary fibrosis-related mutant SP-A2 protein in part through effects on GRP78
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description
researcher
@en
name
Yong-yu Wang
@en
Yong-yu Wang
@nl
type
label
Yong-yu Wang
@en
Yong-yu Wang
@nl
prefLabel
Yong-yu Wang
@en
Yong-yu Wang
@nl
P106
P31
P496
0000-0002-6088-8887