about
Modulation of anxiety through blockade of anandamide hydrolysisSatiety factor oleoylethanolamide recruits the brain histaminergic system to inhibit food intakeRole of bed nucleus of the stria terminalis corticotrophin-releasing factor receptors in frustration stress-induced binge-like palatable food consumption in female rats with a history of food restriction.An anorexic lipid mediator regulated by feeding.Monoaminergic changes in locus coeruleus and dorsal raphe nucleus following noradrenaline depletion.Modulation of meal pattern in the rat by the anorexic lipid mediator oleoylethanolamide.Evaluation of the emotional phenotype and serotonergic neurotransmission of fatty acid amide hydrolase-deficient miceSympathetic activity controls fat-induced oleoylethanolamide signaling in small intestine.Depressive-like behavior is paired to monoaminergic alteration in a murine model of Alzheimer's disease.Effect of glucosamine and its peptidyl-derivative on the production of extracellular matrix components by human primary chondrocytes.Food intake regulates oleoylethanolamide formation and degradation in the proximal small intestineThe lipid messenger OEA links dietary fat intake to satiety.Short- and long-term consequences of prenatal exposure to the cannabinoid agonist WIN55,212-2 on rat glutamate transmission and cognitive functions.Oleoylethanolamide: a novel potential pharmacological alternative to cannabinoid antagonists for the control of appetiteThe role of endocannabinoid signaling in the molecular mechanisms of neurodegeneration in Alzheimer's disease.Endocannabinoid signaling and food addiction.Central mechanisms mediating the hypophagic effects of oleoylethanolamide and N-acylphosphatidylethanolamines: different lipid signals?5-S-cysteinyldopamine neurotoxicity: Influence on the expression of α-synuclein and ERp57 in cellular and animal models of Parkinson's disease.Oleoylethanolamide inhibits food intake in free-feeding rats after oral administration.Preclinical and clinical issues in Alzheimer's disease drug research and development.Glutamatergic alterations and mitochondrial impairment in a murine model of Alzheimer disease.Cold exposure stimulates synthesis of the bioactive lipid oleoylethanolamide in rat adipose tissue.Genetic factors involved in the effects of developmental low-level alcohol induced behavioral alterations in rats.Pharmacological characterization of hydrolysis-resistant analogs of oleoylethanolamide with potent anorexiant properties.Anandamide hydrolysis: a new target for anti-anxiety drugs?Characterization of the fatty acid amide hydrolase inhibitor cyclohexyl carbamic acid 3'-carbamoyl-biphenyl-3-yl ester (URB597): effects on anandamide and oleoylethanolamide deactivation.Altered expression of the CB1 cannabinoid receptor in the triple transgenic mouse model of Alzheimer's disease.Drug context differently regulates cocaine versus heroin self-administration and cocaine- versus heroin-induced Fos mRNA expression in the rat.Developmental exposure to methylmercury elicits early cell death in the cerebral cortex and long-term memory deficits in the rat.Oleoylethanolamide, an endogenous PPAR-alpha agonist, lowers body weight and hyperlipidemia in obese rats.Effects of perinatal exposure to delta-9-tetrahydrocannabinol on the emotional reactivity of the offspring: a longitudinal behavioral study in Wistar rats.Prenatal exposure to the cannabinoid receptor agonist WIN 55,212-2 increases glutamate uptake through overexpression of GLT1 and EAAC1 glutamate transporter subtypes in rat frontal cerebral cortex.Perinatal exposure to delta-9-tetrahydrocannabinol causes enduring cognitive deficits associated with alteration of cortical gene expression and neurotransmission in rats.Chronic agomelatine treatment corrects the abnormalities in the circadian rhythm of motor activity and sleep/wake cycle induced by prenatal restraint stress in adult rats.WAG/Rij rats show a reduced expression of CB₁ receptors in thalamic nuclei and respond to the CB₁ receptor agonist, R(+)WIN55,212-2, with a reduced incidence of spike-wave discharges.Inhibition of anandamide hydrolysis enhances noradrenergic and GABAergic transmission in the prefrontal cortex and basolateral amygdala of rats subjected to acute swim stress.Role of the basolateral amygdala in mediating the effects of the fatty acid amide hydrolase inhibitor URB597 on HPA axis response to stress.High dietary fat intake influences the activation of specific hindbrain and hypothalamic nuclei by the satiety factor oleoylethanolamide.In vivo neurochemical effects of the acetylcholinesterase inhibitor ENA713 in rat hippocampus.Hindbrain noradrenergic input to the hypothalamic PVN mediates the activation of oxytocinergic neurons induced by the satiety factor oleoylethanolamide
P50
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P50
description
researcher
@en
wetenschapper
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name
Silvana Gaetani
@en
Silvana Gaetani
@nl
type
label
Silvana Gaetani
@en
Silvana Gaetani
@nl
prefLabel
Silvana Gaetani
@en
Silvana Gaetani
@nl
P106
P31
P496
0000-0003-2335-7021