about
Molecular identification and expression analysis of filaggrin-2, a member of the S100 fused-type protein familyIsolation of SPINK6 in human skin: selective inhibitor of kallikrein-related peptidasesThe solution structure of the kallikrein-related peptidases inhibitor SPINK6Mucoid Pseudomonas aeruginosa, TNF-alpha, and IL-1beta, but not IL-6, induce human beta-defensin-2 in respiratory epitheliaHuman beta-defensin 3 mediates tissue remodeling processes in articular cartilage by increasing levels of metalloproteinases and reducing levels of their endogenous inhibitorsCharacterization of Spink6 in mouse skin: the conserved inhibitor of kallikrein-related peptidases is reduced by barrier injuryIdentification of lympho-epithelial Kazal-type inhibitor 2 in human skin as a kallikrein-related peptidase 5-specific protease inhibitorKallikreins - The melting pot of activity and functionCell-mediated reduction of human β-defensin 1: a major role for mucosal thioredoxinFocal targeting by human β-defensin 2 disrupts localized virulence factor assembly sites in Enterococcus faecalis.Neutrophil-derived serine proteases modulate innate immune responses.Reddish, scaly, and itchy: how proteases and their inhibitors contribute to inflammatory skin diseases.Human leukocyte elastase induces keratinocyte proliferation by epidermal growth factor receptor activation.Trypsin induces epidermal proliferation and inflammation in murine skin.Immunomodulatory effects of vitamin D on innate and adaptive immune responses to Streptococcus pneumoniae.Staphylococcus aureus subverts cutaneous defense by D-alanylation of teichoic acids.Myeloid differentiation factor 88-dependent signalling controls bacterial growth during colonization and systemic pneumococcal disease in mice.Identification of trichohyalin-like 1, an s100 fused-type protein selectively expressed in hair follicles.The serine protease inhibitor of Kazal-type 7 (SPINK7) is expressed in human skin.Inhibition of kallikrein-related peptidases by the serine protease inhibitor of Kazal-type 6.Enhanced expression and secretion of antimicrobial peptides in atopic dermatitis and after superficial skin injury.Murine filaggrin-2 is involved in epithelial barrier function and down-regulated in metabolically induced skin barrier dysfunction.Differential expression of antimicrobial peptides in margins of chronic wounds.The serine protease inhibitor of Kazal-type 9 (SPINK9) is expressed in lichen simplex chronicus, actinic keratosis and squamous cell carcinoma.Exanthema caused by ingestion of potassium p-aminobenzoate (POTABA-Glenwood(®) ).The antimicrobial protein psoriasin (S100A7) is upregulated in atopic dermatitis and after experimental skin barrier disruption.Decreased Susceptibility of Staphylococcus aureus Small-Colony Variants toward Human Antimicrobial PeptidesSusceptibility of Staphylococcus aureus bacteremia strains to different skin-derived antimicrobial proteinsIdentification of heparin/heparan sulfate interacting protein as a major broad-spectrum antimicrobial protein in lung and small intestineExpression of human beta-defensin-2 and -3 in verrucae vulgares and condylomata acuminataHighly complex peptide aggregates of the S100 fused-type protein hornerin are present in human skinInnate immunity in the skin[Antimicrobial peptides in atopic dermatitis. A paradigm shift?]Increased expression of human beta-defensin 3 in mollusca contagiosumRegulation of kallikrein-related peptidases in the skin - from physiology to diseases to therapeutic optionsAllergic contact dermatitis caused by betulin-containing triterpene extract from the outer bark of birch (Betula alba)SPINK9 stimulates metalloprotease/EGFR-dependent keratinocyte migration via purinergic receptor activation
P50
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P50
description
researcher
@en
wetenschapper
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name
Meyer-Hoffert U
@nl
Ulf Meyer-Hoffert
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Meyer-Hoffert U
@nl
Ulf Meyer-Hoffert
@en
altLabel
Meyer-Hoffert U
@en
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Meyer-Hoffert U
@nl
Ulf Meyer-Hoffert
@en
P106
P31
P496
0000-0003-2305-1428