about
DNA encoding individual mycobacterial antigens protects mice against tuberculosis.Comparison of different delivery systems of DNA vaccination for the induction of protection against tuberculosis in mice and guinea pigs.New strategy for testing efficacy of immunotherapeutic compounds for diabetes in vitro.Improve protective efficacy of a TB DNA-HSP65 vaccine by BCG priming.Detrimental Effect of Fungal 60-kDa Heat Shock Protein on Experimental Paracoccidioides brasiliensis Infection.DNA vaccine containing the mycobacterial hsp65 gene prevented insulitis in MLD-STZ diabetes.Mycobacterium tuberculosis culture filtrate proteins plus CpG Oligodeoxynucleotides confer protection to Mycobacterium bovis BCG-primed mice by inhibiting interleukin-4 secretion.M2 macrophages or IL-33 treatment attenuate ongoing Mycobacterium tuberculosis infection.Role of trehalose dimycolate in recruitment of cells and modulation of production of cytokines and NO in tuberculosis.Systemic Immunological changes in patients with distinct clinical outcomes during Mycobacterium tuberculosis infection.Genetic background affects the expansion of macrophage subsets in the lungs of Mycobacterium tuberculosis-infected hosts.CD11c(+) CD103(+) cells of Mycobacterium tuberculosis-infected C57BL/6 but not of BALB/c mice induce a high frequency of interferon-γ- or interleukin-17-producing CD4(+) cells.Immunotherapy with plasmid DNA encoding mycobacterial hsp65 in association with chemotherapy is a more rapid and efficient form of treatment for tuberculosis in mice.Leukotrienes are not essential for the efficacy of a heterologous vaccine against Mycobacterium tuberculosis infection.B-lymphocytes in bone marrow or lymph nodes can take up plasmid DNA after intramuscular delivery.Host genetic background affects regulatory T-cell activity that influences the magnitude of cellular immune response against Mycobacterium tuberculosis.Oral administration of Saccharomyces cerevisiae UFMG A-905 prevents allergic asthma in mice.Immunosuppressive evidence of Tityus serrulatus toxins Ts6 and Ts15: insights of a novel K(+) channel pattern in T cells.Attenuation of experimental asthma by mycobacterial protein combined with CpG requires a TLR9-dependent IFN-γ-CCR2 signalling circuit.Requirement of MyD88 and Fas pathways for the efficacy of allergen-free immunotherapy.Therapy of tuberculosis in mice by DNA vaccination.Comprehensive gene expression profiling in lungs of mice infected with Mycobacterium tuberculosis following DNAhsp65 immunotherapy.Protective efficacy of different strategies employingMycobacterium lepraeheat-shock protein 65 against tuberculosisCCR4-dependent reduction in the number and suppressor function of CD4Foxp3 cells augments IFN-γ-mediated pulmonary inflammation and aggravates tuberculosis pathogenesisComparison of different delivery systems of vaccination for the induction of protection against tuberculosis in miceGenetic aspects and microenvironment affect expression of CD18 and VLA-4 in experimental tuberculosisThimet oligopeptidase (EC 3.4.24.15), a novel protein on the route of MHC class I antigen presentationHistoplasma capsulatum inhibits apoptosis and Mac-1 expression in leucocytesDownmodulation of CD18 and CD86 on macrophages and VLA-4 on lymphocytes in experimental tuberculosisIncreased levels of interferon-gamma primed by culture filtrate proteins antigen and CpG-ODN immunization do not confer significant protection against Mycobacterium tuberculosis infectionImmune regulatory effect of pHSP65 DNA therapy in pulmonary tuberculosis: activation of CD8+ cells, interferon-gamma recovery and reduction of lung injuryImmune modulation induced by tuberculosis DNA vaccine protects non-obese diabetic mice from diabetes progressionImmunomodulation and protection induced by DNA-hsp65 vaccination in an animal model of arthritisIFN-γ-mediated efficacy of allergen-free immunotherapy using mycobacterial antigens and CpG-ODNProtection conferred by heterologous vaccination against tuberculosis is dependent on the ratio of CD4(+) /CD4(+) Foxp3(+) cellsImprovement of the resistance against early Mycobacterium tuberculosis-infection in the absence of PI3Kγ enzyme is associated with increase of CD4+IL-17+ cells and neutrophilsMycobacterium tuberculosis-infected alveolar epithelial cells modulate dendritic cell function through the HIF-1α-NOS2 axis
P50
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P50
description
researcher
@en
wetenschapper
@nl
name
V L Bonato
@en
V L Bonato
@nl
type
label
V L Bonato
@en
V L Bonato
@nl
prefLabel
V L Bonato
@en
V L Bonato
@nl
P106
P31
P496
0000-0003-4189-2685