Pancreatic cancer foiled by a switch of tumour subtype

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Pancreatic cancer foiled by a switch of tumour subtype

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journal article; published in Nature on 2018-05-14

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journal article; published in Nature on 2018-05-14

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wetenschappelijk artikel

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Pancreatic cancer foiled by a switch of tumour subtype

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Pancreatic cancer foiled by a switch of tumour subtype

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Pancreatic cancer foiled by a switch of tumour subtype

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Pancreatic cancer foiled by a switch of tumour subtype

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Pancreatic cancer foiled by a switch of tumour subtype

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Pancreatic cancer foiled by a switch of tumour subtype

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Pancreatic cancer foiled by a switch of tumour subtype

@en

Pancreatic cancer foiled by a switch of tumour subtype

@en-gb

Pancreatic cancer foiled by a switch of tumour subtype

@nl

P1476

Pancreatic cancer foiled by a switch of tumour subtype

@en

P2093

Fieke Froeling

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P2860

The mechanisms behind the therapeutic activity of BET bromodomain inhibition

PD-1 Blockade in Tumors with Mismatch-Repair Deficiency

Whole genomes redefine the mutational landscape of pancreatic cancer.

Virtual microdissection identifies distinct tumor- and stroma-specific subtypes of pancreatic ductal adenocarcinoma.

Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based therapy for pancreatic ductal adenocarcinoma

Subtypes of pancreatic ductal adenocarcinoma and their differing responses to therapy

Enhancer Reprogramming Promotes Pancreatic Cancer Metastasis.

Genomic analyses identify molecular subtypes of pancreatic cancer.

Cancer Statistics, 2017.

Epigenomic reprogramming during pancreatic cancer progression links anabolic glucose metabolism to distant metastasis.

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d41586-018-05129-6

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500-501

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scholarly article

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10.1038/D41586-018-05129-6

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7706

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557

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David Tuveson

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2018-05-01T00:00:00Z

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1103975470

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29777189

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