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Chlorine-induced injury to the airways in miceCD8 positive T cells express IL-17 in patients with chronic obstructive pulmonary disease.IL-22 contributes to TGF-β1-mediated epithelial-mesenchymal transition in asthmatic bronchial epithelial cells.Th17-associated cytokines promote human airway smooth muscle cell proliferation.Upregulation of IL-17A/F from human lung tissue explants with cigarette smoke exposure: implications for COPD.Aryl hydrocarbon receptor-dependent retention of nuclear HuR suppresses cigarette smoke-induced cyclooxygenase-2 expression independent of DNA-binding.Decreased expression of the NF-κB family member RelB in lung fibroblasts from Smokers with and without COPD potentiates cigarette smoke-induced COX-2 expression.Cigarette smoke increases TLR4 and TLR9 expression and induces cytokine production from CD8(+) T cells in chronic obstructive pulmonary disease.Genetic and histologic evidence for autophagy in asthma pathogenesis.Fibroblast-epithelial cell interactions drive epithelial-mesenchymal transition differently in cells from normal and COPD patients.RelB attenuates cigarette smoke extract-induced apoptosis in association with transcriptional regulation of the aryl hydrocarbon receptor.Aryl hydrocarbon receptor (AhR) attenuation of subchronic cigarette smoke-induced pulmonary neutrophilia is associated with retention of nuclear RelB and suppression of intercellular adhesion molecule-1 (ICAM-1).Aryl hydrocarbon receptor-dependent regulation of miR-196a expression controls lung fibroblast apoptosis but not proliferation.Nitration of respiratory epithelial cells by myeloperoxidase depends on extracellular nitrite.Meniere's disease may be caused by common intraosseous dental pathology--Diagnosis using the comparative compression sign.Nitric oxide and collagen expression in allergic upper-airway disease.T-cell proliferation induced by local application of LPS in the nasal mucosa of nonatopic children.Low levels of the AhR in chronic obstructive pulmonary disease (COPD)-derived lung cells increases COX-2 protein by altering mRNA stability.Differential roles of CXCL2 and CXCL3 and their receptors in regulating normal and asthmatic airway smooth muscle cell migration.The NF-κB family member RelB regulates microRNA miR-146a to suppress cigarette smoke-induced COX-2 protein expression in lung fibroblasts.IL-8 production in response to cigarette smoke is decreased in epithelial cells from COPD patients.Involvement of the cysteinyl-leukotrienes in allergen-induced airway eosinophilia and hyperresponsiveness in the mouse.The dental region constitutes a major blind spot in clinical medicine.Inhibition of allergic airways inflammation and airway hyperresponsiveness in mice by dexamethasone: role of eosinophils, IL-5, eotaxin, and IL-13.Induction of nitric oxide synthase expression by lipopolysaccharide is mediated by calcium-dependent PKCα-β1 in alveolar epithelial cells.Protein nitration in chronic sinusitis and nasal polyposis: role of eosinophils.A role for endoglin in coupling eNOS activity and regulating vascular tone revealed in hereditary hemorrhagic telangiectasia.NOS 1 is required for allergen-induced expression of NOS 2 in mice.The aryl hydrocarbon receptor suppresses cigarette-smoke-induced oxidative stress in association with dioxin response element (DRE)-independent regulation of sulfiredoxin 1.Role of toll-like receptor 4 in protection by bacterial lipopolysaccharide in the nasal mucosa of atopic children but not adults.The use and misuse of Penh in animal models of lung disease.Harvesting airway surface liquid: a comparison of two techniques.Interleukin-17A and vascular remodeling in severe asthma; lack of evidence for a direct role.Influence of gender and interleukin-10 deficiency on the inflammatory response during lung infection with Pseudomonas aeruginosa in mice.CXCL1 inhibits airway smooth muscle cell migration through the decoy receptor Duffy antigen receptor for chemokines.Autocrine-regulated airway smooth muscle cell migration is dependent on IL-17-induced growth-related oncogenes.AB021. Validation of real-world, non-research thoracic CT scans for quantitative analysis of COPD.Are leukocytes in asthmatic patients aging faster? A study of telomere length and disease severity.Ten Principles for More Conservative, Care-Full DiagnosisIL-17A and IL-17F Expression in B Lymphocytes
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P50
name
David H. Eidelman
@en
David H. Eidelman
@nl
type
label
David H. Eidelman
@en
David H. Eidelman
@nl
prefLabel
David H. Eidelman
@en
David H. Eidelman
@nl