about
The human neutrophil subsets defined by the presence or absence of OLFM4 both transmigrate into tissue in vivo and give rise to distinct NETs in vitroCytochalasin B triggers a novel pertussis toxin sensitive pathway in TNF-alpha primed neutrophilsMice chronically fed high-fat diet have increased mortality and disturbed immune response in sepsisEndogenous Acute Phase Serum Amyloid A Lacks Pro-Inflammatory Activity, Contrasting the Two Recombinant Variants That Activate Human Neutrophils through Different ReceptorsA proinflammatory peptide from Helicobacter pylori activates monocytes to induce lymphocyte dysfunction and apoptosisProinflammatory activity of a cecropin-like antibacterial peptide from Helicobacter pylori.Lipopolysaccharide-induced granule mobilization and priming of the neutrophil response to Helicobacter pylori peptide Hp(2-20), which activates formyl peptide receptor-like 1.IRAK-4 mutation (Q293X): rapid detection and characterization of defective post-transcriptional TLR/IL-1R responses in human myeloid and non-myeloid cells.Exopolysaccharides from Burkholderia cenocepacia inhibit neutrophil chemotaxis and scavenge reactive oxygen species.Reactivation of desensitized formyl peptide receptors by platelet activating factor: a novel receptor cross talk mechanism regulating neutrophil superoxide anion production.A pepducin derived from the third intracellular loop of FPR2 is a partial agonist for direct activation of this receptor in neutrophils but a full agonist for cross-talk triggered reactivation of FPR2.Ligand recognition and activation of formyl peptide receptors in neutrophils.Neutrophils from patients with SAPHO syndrome show no signs of aberrant NADPH oxidase-dependent production of intracellular reactive oxygen species.Intracellular generation of superoxide by the phagocyte NADPH oxidase: how, where, and what for?Collection of in vivo transmigrated neutrophils from human skin.A simple skin blister technique for the study of in vivo transmigration of human leukocytes.Phagocyte interactions with Mycobacterium tuberculosis--Simultaneous analysis of phagocytosis, phagosome maturation and intracellular replication by imaging flow cytometry.Antibacterial activity of pepducins, allosterical modulators of formyl peptide receptor signaling.Phenol-Soluble Modulin α Peptide Toxins from Aggressive Staphylococcus aureus Induce Rapid Formation of Neutrophil Extracellular Traps through a Reactive Oxygen Species-Independent Pathway.The leukocyte chemotactic receptor FPR2, but not the closely related FPR1, is sensitive to cell-penetrating pepducins with amino acid sequences descending from the third intracellular receptor loop.A methodological approach to studies of desensitization of the formyl peptide receptor: Role of the read out system, reactive oxygen species and the specific agonist used to trigger neutrophils.Subinhibitory concentrations of the deformylase inhibitor actinonin increase bacterial release of neutrophil-activating peptides: a new approach to antimicrobial chemotherapy.A pepducin designed to modulate P2Y2R function interacts with FPR2 in human neutrophils and transfers ATP to an NADPH-oxidase-activating ligand through a receptor cross-talk mechanism.Quantification of heterotypic granule fusion in human neutrophils by imaging flow cytometry.Serum amyloid A mediates human neutrophil production of reactive oxygen species through a receptor independent of formyl peptide receptor like-1.Increased intracellular oxygen radical production in neutrophils during febrile episodes of periodic fever, aphthous stomatitis, pharyngitis, and cervical adenitis syndrome.Neutrophil NET formation is regulated from the inside by myeloperoxidase-processed reactive oxygen species.Staphylokinase Control of Staphylococcus aureus Biofilm Formation and Detachment Through Host Plasminogen Activation.The Neutrophil Response Induced by an Agonist for Free Fatty Acid Receptor 2 (GPR43) Is Primed by Tumor Necrosis Factor Alpha and by Receptor Uncoupling from the Cytoskeleton but Attenuated by Tissue Recruitment.CFP-10 from Mycobacterium tuberculosis selectively activates human neutrophils through a pertussis toxin-sensitive chemotactic receptor.The host defense peptide LL-37 selectively permeabilizes apoptotic leukocytes.Receptor-dependent and -independent immunomodulatory effects of phenol-soluble modulin peptides from Staphylococcus aureus on human neutrophils are abrogated through peptide inactivation by reactive oxygen species.Cord-forming mycobacteria induce DNA meshwork formation by human peripheral blood mononuclear cells.Formyl peptide derived lipopeptides disclose differences between the receptors in mouse and men and call the pepducin concept in questionInterleukin-8-derived peptide has antibacterial activity.Divergent effects on phagocytosis by macrophage-derived oxygen radicals.Immunostimulatory DNA induces degranulation and NADPH-oxidase activation in human neutrophils while concomitantly inhibiting chemotaxis and phagocytosis.NADPH-oxidase activation in murine neutrophils via formyl peptide receptors.Reactivation of formyl peptide receptors triggers the neutrophil NADPH-oxidase but not a transient rise in intracellular calcium.Outer membrane protein A deficient Escherichia coli activates neutrophils to produce superoxide and shows increased susceptibility to antibacterial peptides.
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description
Swedish researcher at University of Gothenburg
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Zweeds onderzoeker
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svensk forsker
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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Johan Bylund
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P2038
Johan_Bylund
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P496
0000-0002-9094-6478