about
Using measures of single-cell physiology and physiological state to understand organismic agingA role for autophagy in the extension of lifespan by dietary restriction in C. elegansThe core apoptotic executioner proteins CED-3 and CED-4 promote initiation of neuronal regeneration in Caenorhabditis elegansDeletion of microRNA-80 activates dietary restriction to extend C. elegans healthspan and lifespanCeleST: computer vision software for quantitative analysis of C. elegans swim behavior reveals novel features of locomotionMolecular modeling of mechanotransduction in the nematode Caenorhabditis elegansMetformin induces a dietary restriction-like state and the oxidative stress response to extend C. elegans Healthspan via AMPK, LKB1, and SKN-1Sequence Relationships among C. elegans, D. melanogaster and Human microRNAs Highlight the Extensive Conservation of microRNAs in BiologyNecrotic cell death in C. elegans requires the function of calreticulin and regulators of Ca(2+) release from the endoplasmic reticulum.Specific aspartyl and calpain proteases are required for neurodegeneration in C. elegans.Dramatic fertility decline in aging C. elegans males is associated with mating execution deficits rather than diminished sperm quality.Specific roles for DEG/ENaC and TRP channels in touch and thermosensation in C. elegans nociceptors.unc-8, a DEG/ENaC family member, encodes a subunit of a candidate mechanically gated channel that modulates C. elegans locomotion.Spatial asymmetry in the mechanosensory phenotypes of the C. elegans DEG/ENaC gene mec-10.Caloric restriction and lifespan: a role for protein turnover?Mechanotransduction in Caenorhabditis elegans: the role of DEG/ENaC ion channels.Protons at the gate: DEG/ENaC ion channels help us feel and remember.Neuronal repair: Apoptotic proteins make good.Dying for a cause: invertebrate genetics takes on human neurodegeneration.UNC-4/UNC-37-dependent repression of motor neuron-specific genes controls synaptic choice in Caenorhabditis elegans.Neurite sprouting and synapse deterioration in the aging Caenorhabditis elegans nervous systemGenetic and molecular analysis of a Caenorhabditis elegans beta-tubulin that conveys benzimidazole sensitivity.Sequence and transmembrane topology of MEC-4, an ion channel subunit required for mechanotransduction in Caenorhabditis elegans.Single swim sessions in C. elegans induce key features of mammalian exercise.Age-Related Phasic Patterns of Mitochondrial Maintenance in Adult Caenorhabditis elegans Neurons.Genetically targeted cell disruption in Caenorhabditis elegansComplex expression dynamics and robustness in C. elegans insulin networks.A glial DEG/ENaC channel functions with neuronal channel DEG-1 to mediate specific sensory functions in C. elegansCulture of embryonic C. elegans cells for electrophysiological and pharmacological analyses.Heterologous expression of C. elegans ion channels in Xenopus oocytes.Noncanonical cell death programs in the nematode Caenorhabditis elegans.FOXO3a is broadly neuroprotective in vitro and in vivo against insults implicated in motor neuron diseases.MicroRNAs in C. elegans Aging: Molecular Insurance for Robustness?NRA-2, a nicalin homolog, regulates neuronal death by controlling surface localization of toxic Caenorhabditis elegans DEG/ENaC channels.EGF signaling comes of age: promotion of healthy aging in C. elegansA long journey to reproducible results.Automated Analysis of C. elegans Swim Behavior Using CeleST Software.C. elegans neurons jettison protein aggregates and mitochondria under neurotoxic stressNovel EGF pathway regulators modulate C. elegans healthspan and lifespan via EGF receptor, PLC-gamma, and IP3R activation.Mechanism of longevity extension of Caenorhabditis elegans induced by pentagalloyl glucose isolated from eucalyptus leaves.
P50
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P50
description
biologist
@en
name
Monica Driscoll
@en
Monica Driscoll
@nl
type
label
Monica Driscoll
@en
Monica Driscoll
@nl
altLabel
Driscoll M
@en
Driscoll M.
@en
Driscoll
@en
M Driscoll
@en
M. Driscoll
@en
prefLabel
Monica Driscoll
@en
Monica Driscoll
@nl