about
Single-cell molecular analysis defines therapy response and immunophenotype of stem cell subpopulations in CML.Tapasin facilitation of natural HLA-A and -B allomorphs is strongly influenced by peptide length, depends on stability, and separates closely related allomorphs.Tapasin discriminates peptide-human leukocyte antigen-A*02:01 complexes formed with natural ligands.Tapasin and human leukocyte antigen class I dysregulation correlates with survival in glioblastoma multiforme.Viral proteins interfering with antigen presentation target the major histocompatibility complex class I peptide-loading complex.HLA class I is most tightly linked to levels of tapasin compared with other antigen-processing proteins in glioblastoma.Expression of matrix metalloprotease-2, -7 and -9 on human colon, liver and bile duct cell lines by enteric and gastric Helicobacter species.HLA class I is most tightly linked to levels of tapasin compared with other antigen-processing proteins in glioblastoma.The outermost N-terminal region of tapasin facilitates folding of major histocompatibility complex class I.
P50
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P50
name
Linda Geironson
@en
Linda Geironson
@nl
type
label
Linda Geironson
@en
Linda Geironson
@nl
prefLabel
Linda Geironson
@en
Linda Geironson
@nl