about
Notch2 signaling sensitizes endothelial cells to apoptosis by negatively regulating the key protective molecule survivin.A combination of in silico and SAR studies to identify binding hot spots of Bcl-xL inhibitors.Bcl-xL controls a switch between cell death modes during mitotic arrest(13)C and (15)N natural isotope abundance reflects breast cancer cell metabolism.A Ring-D-Seco-Tetranortriterpenoid from Seeds of Carapa procera Active against Breast Cancer Cell Lines.Novel 1,6-naphthyridin-2(1H)-ones as potential anticancer agents targeting Hsp90.Preliminary Studies on the Activity of Mixed Polyphenol-Heterocyclic Systems Against B16-F10 Melanoma Cancer Cells.Phytochemicals isolated from leaves of Chromolaena odorata: impact on viability and clonogenicity of cancer cell lines.Impact of XIAP protein levels on the survival of myeloma cellsCarcinoma-associated fucosylated antigens are markers of the epithelial state and can contribute to cell adhesion through CLEC17A (Prolectin).Canonical nuclear factor kappaB pathway inhibition blocks myeloma cell growth and induces apoptosis in strong synergy with TRAIL.Significant impact of survivin on myeloma cell growth.BCL-XL directly modulates RAS signalling to favour cancer cell stemness.E2F1 interacts with BCL-xL and regulates its subcellular localization dynamics to trigger cell death.Cell cycle and cell death associates in western France.p53 regulates CD46 expression and measles virus infection in myeloma cells[Survivin in cancerology : molecular aspects and therapeutic applications]Design, synthesis and biological evaluation of new inhibitors of Bax/Bcl-xL interaction in cancer cellsInteractions between cancer-associated fibroblasts and tumor cells promote MCL-1 dependency in estrogen receptor-positive breast cancersEpinephrine Infiltration of Adipose Tissue Impacts MCF7 Breast Cancer Cells and Total Lipid ContentSTING-dependent paracriny shapes apoptotic priming of breast tumors in response to anti-mitotic treatmentTargeting PUMA/Bcl-xL interaction by new specific compounds to unleash apoptotic process in cancer cellsMitotic stress-induced secretome primes cancer cells to apoptosis and maximizes paclitaxel response in breast tumors when combined with BCL-xL-targeting BH3 mimetics
P50
Q33518506-B7307732-61B0-467C-BB8C-2CEBE5273A06Q35583314-087C1760-3674-46B9-8D17-6621AD6E5F67Q36179601-F9744195-F63F-4B17-99EA-91EFB4D04196Q37291288-8EEA96F9-7E95-48D2-8F9D-65496BD4990BQ38768506-3CD7B87A-0393-4309-BAAD-7571EA8095DDQ38772750-329A379B-43EC-40C0-A3A0-DDC75B643B50Q38798249-F8AB6F06-CEC4-43A8-B6D4-73C62A2C644FQ39296212-04878980-F639-4182-8014-1CE97A42498BQ39918873-66735A45-5996-4D47-A2BE-F1D2FB073B31Q39974354-5AE830A0-1084-4962-8AFF-BB065B4B3965Q40065651-4EA52CA4-E753-4779-8F31-E66FAB0F6860Q40168320-D9C0ED97-426F-42E5-AE5D-0787DEB0F525Q42639369-00131563-873C-4795-B618-4BD1ADB73C8FQ47301301-5420B3E7-FC40-4EB1-828C-E7EA6335867DQ53573521-C44BF75A-70D7-449C-8040-BE5AC90F1CDFQ59789486-D720F10B-E2A2-4B88-BC51-8A20B8011F84Q79701757-FE293921-DF72-4CBB-8C70-E379EBE087F2Q87410418-3A7A8844-39A4-4F0E-9192-915206E39556Q90989910-44F0BFDA-7BD4-4D55-9574-B2EA9A3230F7Q91254616-49184CB1-7736-4BF5-8468-6EB83E7FD384Q92642973-A6505CAD-80D4-4E52-9124-73A4A969037AQ93214065-FF428705-905C-469F-96D0-D77674C5C5F3Q94547835-8523A618-03DC-4ACE-A62F-FE1F0F4EE085
P50
description
researcher ORCID ID=0000-0001-5171-9937
@en
wetenschapper
@nl
name
Sophie Barillé-Nion
@en
Sophie Barillé-Nion
@nl
type
label
Sophie Barillé-Nion
@en
Sophie Barillé-Nion
@nl
prefLabel
Sophie Barillé-Nion
@en
Sophie Barillé-Nion
@nl
P106
P31
P496
0000-0001-5171-9937