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Polysialic acid is required for dopamine D2 receptor-mediated plasticity involving inhibitory circuits of the rat medial prefrontal cortexDifferential evolution of PSA-NCAM expression during aging of the rat telencephalonChronic antidepressant treatment induces contrasting patterns of synaptophysin and PSA-NCAM expression in different regions of the adult rat telencephalonEarly Social Isolation Stress and Perinatal NMDA Receptor Antagonist Treatment Induce Changes in the Structure and Neurochemistry of Inhibitory Neurons of the Adult Amygdala and Prefrontal Cortex.Long-term behavioral programming induced by peripuberty stress in rats is accompanied by GABAergic-related alterations in the Amygdala.Polysialic Acid Acute Depletion Induces Structural Plasticity in Interneurons and Impairs the Excitation/Inhibition Balance in Medial Prefrontal Cortex Organotypic Cultures.Reduced interneuronal dendritic arborization in CA1 but not in CA3 region of mice subjected to chronic mild stress.Structural plasticity of interneurons in the adult brain: role of PSA-NCAM and implications for psychiatric disorders.Effects of Chronic Dopamine D2R Agonist Treatment and Polysialic Acid Depletion on Dendritic Spine Density and Excitatory Neurotransmission in the mPFC of Adult Rats.The brain as immunoprecipitator of serum autoantibodies against N-Methyl-D-aspartate receptor subunit NR1.Dopamine acting through D2 receptors modulates the expression of PSA-NCAM, a molecule related to neuronal structural plasticity, in the medial prefrontal cortex of adult rats.A population of prenatally generated cells in the rat paleocortex maintains an immature neuronal phenotype into adulthood.The polysialylated form of the neural cell adhesion molecule (PSA-NCAM) is expressed in a subpopulation of mature cortical interneurons characterized by reduced structural features and connectivity.Response to Letter Regarding Article, "Preexisting Serum Autoantibodies Against the NMDAR Subunit NR1 Modulate Evolution of Lesion Size in Acute Ischemic Stroke".Streptozotocin diabetic mice display depressive-like behavior and alterations in the structure, neurotransmission and plasticity of medial prefrontal cortex interneurons.PSA-NCAM expression in the human prefrontal cortex.N-methyl-d-aspartate receptor expression during adult neurogenesis in the rat dentate gyrus.Chronic fluoxetine treatment increases the expression of PSA-NCAM in the medial prefrontal cortex.All naturally occurring autoantibodies against the NMDA receptor subunit NR1 have pathogenic potential irrespective of epitope and immunoglobulin class.Chronic stress induces changes in the structure of interneurons and in the expression of molecules related to neuronal structural plasticity and inhibitory neurotransmission in the amygdala of adult mice.The dendritic spines of interneurons are dynamic structures influenced by PSA-NCAM expression.Automated analysis of images for molecular quantification in immunohistochemistry.
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description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Esther Castillo-Gómez
@en
Esther Castillo-Gómez
@nl
type
label
Esther Castillo-Gómez
@en
Esther Castillo-Gómez
@nl
altLabel
Castillo-Gómez E
@en
prefLabel
Esther Castillo-Gómez
@en
Esther Castillo-Gómez
@nl
P31
P496
0000-0002-7566-0340