about
Aging-related loss of the chromatin protein HMGB2 in articular cartilage is linked to reduced cellularity and osteoarthritisMitochondrial activity is modulated by TNFalpha and IL-1beta in normal human chondrocyte cells.Autophagy is a protective mechanism in normal cartilage, and its aging-related loss is linked with cell death and osteoarthritisMechanical injury suppresses autophagy regulators and pharmacologic activation of autophagy results in chondroprotection.The relationship of autophagy defects to cartilage damage during joint aging in a mouse model.FoxO transcription factors support oxidative stress resistance in human chondrocytesAutophagy activation and protection from mitochondrial dysfunction in human chondrocytes.Autophagy and cartilage homeostasis mechanisms in joint health, aging and OAAutophagy activation by rapamycin reduces severity of experimental osteoarthritis.Expression patterns and function of chromatin protein HMGB2 during mesenchymal stem cell differentiation.Mitochondrial dysfunction activates cyclooxygenase 2 expression in cultured normal human chondrocytes.Compromised autophagy precedes meniscus degeneration and cartilage damage in mice.[Evidence of inflammatory mechanisms in osteoarthritis].Phosphatase-1 and -2A inhibition modulates apoptosis in human osteoarthritis chondrocytes independently of nitric oxide production.Effect of nitric oxide on mitochondrial respiratory activity of human articular chondrocytes.Pig chondrocyte xenoimplants for human chondral defect repair: an in vitro model.Differential effects of tumor necrosis factor-alpha and interleukin-1beta on cell death in human articular chondrocytes.Cytokines, tumor necrosis factor-alpha and interleukin-1beta, differentially regulate apoptosis in osteoarthritis cultured human chondrocytes.Anti-apoptotic effect of transforming growth factor-beta1 on human articular chondrocytes: role of protein phosphatase 2A.Insulin decreases autophagy and leads to cartilage degradation.Role of the Inflammation-Autophagy-Senescence Integrative Network in Osteoarthritis.Autophagy: a new therapeutic target in cartilage injury and osteoarthritisDiabetes-accelerated experimental osteoarthritis is prevented by autophagy activationFibrates as drugs with senolytic and autophagic activity for osteoarthritis therapyExpression patterns and function of chromatin protein HMGB2 during mesenchymal stem cell differentiation
P50
Q28509293-FB771CAF-32AA-40E2-B889-2664C5E2BDFFQ33242572-A7F2DCF6-A3EA-4E8A-9763-20998E8A2ED4Q33729300-EDC365EC-0016-4A15-BBCE-0FBB3FCA03DFQ34228180-B5CFAEE3-FE28-47B3-A259-3153DD411736Q34464179-6B366B0B-72CD-40C6-9049-B38748CE05F5Q34626746-79C222F6-00AD-4FE8-BE7D-CD54F2CBCF37Q35236233-629A141F-C4D3-4D85-AAB9-F2D82CC7BD85Q35335855-E414B5F6-EF97-492C-AC1D-46FC0C4CA009Q35803625-FC073AC4-9C6E-42AE-99DC-8F7BAAB5E45CQ35842175-8F6D4BFE-33D0-4381-BB9E-C4A705D9669AQ42439611-43CD170B-CBB9-4D70-B8D8-51661DCCD8E3Q42513584-3AE19600-B122-4EFF-97D1-538DE1A5097AQ42728772-936938AB-8835-4E13-9D82-A85899458172Q43048614-2E1292F1-4B3E-4B7B-836F-437842FF5E9DQ43049067-B270BF94-810B-4312-918F-16FCD72584D8Q44983937-8F3826DC-3B64-4155-8409-06D4958CC673Q46870280-D2C88B3E-677F-482B-9F24-FB2820C3C2D0Q46955692-23FD7F3B-50F4-486F-A792-BE412B4CB1D1Q50634285-6F9A997C-9EB7-4BA2-8339-7726CCF79B84Q54235231-802C0AEA-E88B-45F2-9D9B-A5CB621C6A79Q55516438-C79A39E3-BCB3-48EE-A393-3A076756631BQ83803840-DBB32DB8-3D5C-4A00-A20C-79FA0226D6C0Q87845924-1DDA9CCA-182C-4403-B248-532055D11238Q91720482-62F558A1-8DBA-4CC8-8C22-E48BC6B105A8Q93553667-B73A346E-BEC4-468F-8EB6-C1BB3FC06383
P50
description
investigador
@es
researcher
@en
wetenschapper
@nl
name
B Caramés
@en
B Caramés
@nl
type
label
B Caramés
@en
B Caramés
@nl
prefLabel
B Caramés
@en
B Caramés
@nl
P31
P496
0000-0002-8353-6526