about
Changes in skeletal muscle mitochondria in response to the development of type 2 diabetes or prevention by daily wheel running in hyperphagic OLETF ratsSkeletal muscle mitochondrial and metabolic responses to a high-fat diet in female rats bred for high and low aerobic capacity.Intrinsic aerobic capacity impacts susceptibility to acute high-fat diet-induced hepatic steatosisTreating NAFLD in OLETF rats with vigorous-intensity interval exercise trainingDaily exercise vs. caloric restriction for prevention of nonalcoholic fatty liver disease in the OLETF rat model.Female rats selectively bred for high intrinsic aerobic fitness are protected from ovariectomy-associated metabolic dysfunctionGut microbiota are linked to increased susceptibility to hepatic steatosis in low-aerobic-capacity rats fed an acute high-fat dietPGC-1α overexpression results in increased hepatic fatty acid oxidation with reduced triacylglycerol accumulation and secretion.Differential effects of low-fat and high-fat diets on fed-state hepatic triacylglycerol secretion, hepatic fatty acid profiles, and DGAT-1 protein expression in obese-prone Sprague-Dawley rats.The presence of the ovary prevents hepatic mitochondrial oxidative stress in young and aged female mice through glutathione peroxidase 1Effects of ovariectomy and intrinsic aerobic capacity on tissue-specific insulin sensitivity.Fibroblast growth factor 21 and exercise-induced hepatic mitochondrial adaptations.Reduced hepatic mitochondrial respiration following acute high-fat diet is prevented by PGC-1α overexpression.Low aerobic capacity and high-fat diet contribute to oxidative stress and IRS-1 degradation in the kidney.Combining metformin and aerobic exercise training in the treatment of type 2 diabetes and NAFLD in OLETF rats.Exercise and Omega-3 Polyunsaturated Fatty Acid Supplementation for the Treatment of Hepatic Steatosis in Hyperphagic OLETF RatsImpact of various exercise modalities on hepatic mitochondrial function.Angiotensin II-induced NADPH oxidase activation impairs insulin signaling in skeletal muscle cells.Voluntary wheel running selectively augments insulin-stimulated vasodilation in arterioles from white skeletal muscle of insulin-resistant rats.Aerobic capacity and hepatic mitochondrial lipid oxidation alters susceptibility for chronic high-fat diet-induced hepatic steatosis.Combining metformin therapy with caloric restriction for the management of type 2 diabetes and nonalcoholic fatty liver disease in obese rats.Deficiency in the Heat Stress Response Could Underlie Susceptibility to Metabolic DiseaseFibroblast growth factor 21 increases hepatic oxidative capacity but not physical activity or energy expenditure in hepatic peroxisome proliferator-activated receptor γ coactivator-1α-deficient mice.Aerobic capacity mediates susceptibility for the transition from steatosis to steatohepatitis.Mineralocorticoid receptor blockade attenuates chronic overexpression of the renin-angiotensin-aldosterone system stimulation of reduced nicotinamide adenine dinucleotide phosphate oxidase and cardiac remodeling.Intrinsic (Genetic) Aerobic Fitness Impacts Susceptibility for Metabolic Disease.Aerobic exercise training in the treatment of non-alcoholic fatty liver disease related fibrosis.Hepatic mitochondrial adaptations to physical activity: impact of sexual dimorphism, PGC1α and BNIP3-mediated mitophagyHeat shock protein 72 regulates hepatic lipid accumulationSex modulates hepatic mitochondrial adaptations to high-fat diet and physical activityeNOS deletion impairs mitochondrial quality control and exacerbates Western diet-induced NASHIntrinsic High Aerobic Capacity in Male Rats Protects Against Diet-Induced Insulin Resistance
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description
investigador
@es
researcher
@en
wetenschapper
@nl
name
E Matthew Morris
@en
E Matthew Morris
@nl
type
label
E Matthew Morris
@en
E Matthew Morris
@nl
prefLabel
E Matthew Morris
@en
E Matthew Morris
@nl
P108
P31
P496
0000-0001-7046-3623