about
Inhibition of endogenous hydrogen sulfide formation reduces the organ injury caused by endotoxemia.Lipoproteins in inflammation and sepsis. I. Basic science.Glycogen synthase kinase 3beta as a target for the therapy of shock and inflammation.Reduction of experimental colitis in the rat by inhibitors of glycogen synthase kinase-3beta.Lysophosphatidylcholine reduces the organ injury and dysfunction in rodent models of gram-negative and gram-positive shock.The PPAR-gamma ligand 15-deoxy(delta12,14) prostaglandin J2 reduces the liver injury in endotoxic shock.Endogenous ligands of PPAR-gamma reduce the liver injury in haemorrhagic shock.Role of peroxisome proliferator-activated receptor-gamma in the protection afforded by 15-deoxydelta12,14 prostaglandin J2 against the multiple organ failure caused by endotoxin.Urocortin does not reduce the renal injury and dysfunction caused by experimental ischaemia/reperfusion.Reduction of the multiple organ injury and dysfunction caused by endotoxemia in 5-lipoxygenase knockout mice and by the 5-lipoxygenase inhibitor zileuton.15d-prostaglandin J2 reduces multiple organ failure caused by wall-fragment of Gram-positive and Gram-negative bacteria.The peroxisome proliferator-activated receptor-gamma ligand 15-deoxyDelta12,14 prostaglandin J2 reduces the organ injury in hemorrhagic shock.The selective PPARgamma antagonist GW9662 reverses the protection of LPS in a model of renal ischemia-reperfusion.GSK-3beta inhibitors attenuate the organ injury/dysfunction caused by endotoxemia in the rat.Peroxisome proliferator-activated receptor-gamma antagonists GW9662 and T0070907 reduce the protective effects of lipopolysaccharide preconditioning against organ failure caused by endotoxemia.Erythropoietin attenuates the tissue injury associated with hemorrhagic shock and myocardial ischemia.Inhibiting glycogen synthase kinase 3beta in sepsis.Effects of Long-Term Intervention with Lactobacillus helveticus- Fermented Milk on Bone Mineral Density and Bone Mineral Content in Growing RatsLysophosphatidic acid reduces the organ injury caused by endotoxemia-a role for G-protein-coupled receptors and peroxisome proliferator-activated receptor-gammaHydrogen sulfide and sulfite: novel mediators in the pathophysiology of shock and inflammation
P50
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P50
name
Marika Collin
@en
type
label
Marika Collin
@en
prefLabel
Marika Collin
@en