about
Inhibition of nuclear factor of activated T-cells (NFAT) suppresses accelerated atherosclerosis in diabetic miceADAMTS-7 is associated with a high-risk plaque phenotype in human atherosclerosis.IL-25 inhibits atherosclerosis development in apolipoprotein E deficient miceLow elastin content of carotid plaques is associated with increased risk of ipsilateral strokeDystrophin deficiency reduces atherosclerotic plaque development in ApoE-null miceHigh levels of cathepsin D and cystatin B are associated with increased risk of coronary events.Sphingolipids Contribute to Human Atherosclerotic Plaque Inflammation.Atheroprotective effects of Alum are associated with capture of oxidized LDL antigens and activation of regulatory T cells.FcgammaRIIB inhibits the development of atherosclerosis in low-density lipoprotein receptor-deficient mice.Plasma S100A8/A9 correlates with blood neutrophil counts, traditional risk factors, and cardiovascular disease in middle-aged healthy individuals.β-Sarcoglycan Deficiency Reduces Atherosclerotic Plaque Development in ApoE-Null Mice.Increased aldehyde-modification of collagen type IV in symptomatic plaques--a possible cause of endothelial dysfunction.Hyperglycemia does not affect tissue repair responses in shear stress-induced atherosclerotic plaques in ApoE-/- mice.Elevated Plasma Levels of MMP-12 Are Associated With Atherosclerotic Burden and Symptomatic Cardiovascular Disease in Subjects With Type 2 DiabetesActivation of immune responses against the basement membrane component collagen type IV does not affect the development of atherosclerosis in ApoE-deficient miceEvidence for a role of regulatory T cells in mediating the atheroprotective effect of apolipoprotein B peptide vaccineImmunization of apoE-/- mice with aldehyde-modified fibronectin inhibits the development of atherosclerosisHigh plasma levels of heparin-binding epidermal growth factor are associated with a more stable plaque phenotype and reduced incidence of coronary eventsImpaired fibrous repair: a possible contributor to atherosclerotic plaque vulnerability in patients with type II diabetesInterleukin-25 (IL-25) has a protective role in atherosclerosis development in the aortic arch in miceActivation of imidazoline receptor I2, and improved pancreatic β-cell function in human isletsOrphan G-protein coupled receptor 183 (GPR183) potentiates insulin secretion and prevents glucotoxicity-induced β-cell dysfunctionAn osteopontin-derived peptide inhibits human hair growth at least in part by decreasing fibroblast growth factor-7 production in outer root sheath keratinocytesCartilage Oligomeric Matrix Protein Associates With a Vulnerable Plaque Phenotype in Human Atherosclerotic Plaques
P50
Q27317653-9E5C36D4-9B04-4702-838E-1755444396F2Q33808002-3AF0A2A4-AC23-4595-9B71-48192B75DE15Q35023076-20BBF4D9-DEC3-41CC-A151-549A7837411EQ35213758-FC1F2E70-514A-40B0-8D0F-9BF98CBBA214Q36034211-DE8AB6E0-A80C-4DED-8F5E-D19E0B71F51FQ36517244-65BFD12C-16E4-450E-AFC5-EDE6CC455D86Q38779976-5EF39007-B4B1-4342-8717-FA73550FD924Q39975880-049447EC-062B-4AD5-A712-D4B2B446EFD8Q43185229-8A52984A-E6F4-4A2C-82A0-05A70651A770Q45020812-2548FCF1-4C4D-49AC-BDA9-6D0F3D6B57F9Q47999301-ADB840CF-A738-4D5E-99E0-ED32B794D74CQ53592350-D052616D-0015-4DD2-8DA7-8DCC4425ED41Q55355643-811A72BD-BB5E-4193-A9F6-4C06E0160863Q57563910-F19866DF-B421-4517-8D78-DAADCF2C79EEQ64052963-47EE4279-E3D6-48EA-BF9A-24E4DAF606F0Q82652342-C5A811EB-6A09-4036-886D-A430C8AA4680Q83865491-18BE865E-D5F1-431A-A0EA-A970D1D786F2Q85893654-8A66C9DD-576F-4763-A3AA-AED7BC83A92CQ87409473-F37C51D7-F700-462B-8DAF-89FE117F86EEQ88152524-CD272404-EB62-4C8D-82F7-59D66F5F7BC1Q89573304-48A0DA67-870E-4A91-8E94-340DEF556E62Q90256054-2DCD72C5-F016-469D-A410-1BCE9E211A8CQ93123009-7EA87F56-F715-4936-8AFC-6F487BA02FFDQ93176698-48E1DF89-163F-45D9-B9EA-B284EF7F3C6A
P50
description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Pontus Dunér
@en
Pontus Dunér
@nl
type
label
Pontus Dunér
@en
Pontus Dunér
@nl
prefLabel
Pontus Dunér
@en
Pontus Dunér
@nl
P31
P496
0000-0003-4171-5646