about
Dimerization of complement factor H-related proteins modulates complement activation in vivoCompetition between antagonistic complement factors for a single protein on N. meningitidis rules disease susceptibilityA hybrid CFHR3-1 gene causes familial C3 glomerulopathy.Factor H-related proteins determine complement-activating surfaces.Elevated factor H-related protein 1 and factor H pathogenic variants decrease complement regulation in IgA nephropathy.Factor H-related protein 5 interacts with pentraxin 3 and the extracellular matrix and modulates complement activation.Factor H Competitor Generated by Gene Conversion Events Associates with Atypical Hemolytic Uremic Syndrome.Common and rare genetic variants of complement components in human disease
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Q27676814-09FD3C3B-ECD1-4974-B84F-1F1BE95D3906Q34753608-03532857-4A82-43E6-BDA3-6055B0083FC4Q36050943-D4248CC6-3557-429C-A807-DEC6C5D06829Q38483777-7E1A7E7E-CE18-4A6C-A732-CAE3D98147B7Q38715148-52F709B5-260D-40C5-94F0-0BEB3DCA89B1Q40161972-03AEBB3C-7826-4B00-93CD-DEEDC1BE4AB2Q47674559-D6B3A97C-281E-4674-A4AA-4BE6A5617802Q89142244-B1627172-B991-43B8-9E35-07DA132FA458
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description
researcher (ORCID 0000-0002-4978-2483)
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name
Elena Goicoechea de Jorge
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type
label
Elena Goicoechea de Jorge
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prefLabel
Elena Goicoechea de Jorge
@en
P31
P496
0000-0002-4978-2483