about
Zika virus: An emergent neuropathological agentPersistence and pathogenesis of the neurotropic polyomavirus JCInhibition of HSV-1 Replication by Gene Editing StrategyInterplay of Rad51 with NF-κB pathway stimulates expression of HIV-1.Rad51 activates polyomavirus JC early transcriptionCRISPR/Cas9 System as an Agent for Eliminating Polyomavirus JC InfectionExpression of Signaling Molecules in Progressive Multifocal LeukoencephalopathyGene Editing for Treatment of Neurological Infections.Epigenetic regulation of polyomavirus JC.The DNA damage response promotes polyomavirus JC infection by nucleus to cytoplasm NF- kappaB activation.Degradation of polyomavirus JC T-antigen by stress involves the LIP isoform of C/EBP.Epigenetic regulation of polyomavirus JC involves acetylation of specific lysine residues in NF-κB p65.Cooperative roles of NF-κB and NFAT4 in polyomavirus JC regulation at the KB control element.Lentiviral transduction of neuronal cells.The Brd4 acetyllysine-binding protein is involved in activation of polyomavirus JC.Discovery and Characterization of Novel Trans-Spliced Products of Human Polyoma JC Virus Late Transcripts from PML Patients.Viral tumor antigen expression is no longer required in radiation-resistant subpopulation of JCV induced mouse medulloblastoma cellsA Broad Application of CRISPR Cas9 in Infectious Diseases of Central Nervous System
P50
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P50
description
investigador
@es
researcher
@en
name
Hassen S Wollebo
@en
type
label
Hassen S Wollebo
@en
prefLabel
Hassen S Wollebo
@en
P31
P496
0000-0003-2285-3496