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Defining the Roles of TcdA and TcdB in Localized Gastrointestinal Disease, Systemic Organ Damage, and the Host Response during Clostridium difficile InfectionsClostridium difficile toxins induce VEGF-A and vascular permeability to promote disease pathogenesisClostridioides difficile infection damages colonic stem cells via TcdB, impairing epithelial repair and recovery from diseaseEnterotoxic Clostridia: Clostridioides difficile InfectionsIn silico, in vitro and in vivo analysis of putative virulence factors identified in large clostridial toxin-negative, binary toxin- producing C. difficile strains
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description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Steven Mileto
@en
Steven Mileto
@nl
type
label
Steven Mileto
@en
Steven Mileto
@nl
prefLabel
Steven Mileto
@en
Steven Mileto
@nl
P108
P31
P496
0000-0003-2599-8957