about
miR-155 Overexpression Promotes Genomic Instability by Reducing High-fidelity Polymerase Delta Expression and Activating Error-Prone DSB RepairIn vivo correction of anaemia in β-thalassemic mice by γPNA-mediated gene editing with nanoparticle delivery2-Hydroxyglutarate produced by neomorphic IDH mutations suppresses homologous recombination and induces PARP inhibitor sensitivity.The cytotoxicity of (-)-lomaiviticin A arises from induction of double-strand breaks in DNA.Silencing of the DNA mismatch repair gene MLH1 induced by hypoxic stress in a pathway dependent on the histone demethylase LSD1LKB1 preserves genome integrity by stimulating BRCA1 expression.Interplay between DNA repair and inflammation, and the link to cancerSingle-stranded γPNAs for in vivo site-specific genome editing via Watson-Crick recognition.Modified poly(lactic-co-glycolic acid) nanoparticles for enhanced cellular uptake and gene editing in the lung.MicroRNA silencing for cancer therapy targeted to the tumour microenvironment.YU238259 Is a Novel Inhibitor of Homology-Dependent DNA Repair That Exhibits Synthetic Lethality and Radiosensitization in Repair-Deficient Tumors.Nanoparticles that deliver triplex-forming peptide nucleic acid molecules correct F508del CFTR in airway epitheliumDNA-dependent targeting of cell nuclei by a lupus autoantibody.Triplex-forming peptide nucleic acids induce heritable elevations in gamma-globin expression in hematopoietic progenitor cells.Mechanism of action studies of lomaiviticin A and the monomeric lomaiviticin aglycon. Selective and potent activity toward DNA double-strand break repair-deficient cell lines.Radiation sensitivity and sensitization in melanoma.Site-specific Genome Editing in PBMCs With PLGA Nanoparticle-delivered PNAs Confers HIV-1 Resistance in Humanized Mice.microRNAs in cancer cell response to ionizing radiation.Triplex-mediated genome targeting and editing.Peptide nucleic acid-mediated recombination for targeted genomic repair and modification.Multifaceted control of DNA repair pathways by the hypoxic tumor microenvironmentNanotechnology for delivery of peptide nucleic acids (PNAs)Nickel induces transcriptional down-regulation of DNA repair pathways in tumorigenic and non-tumorigenic lung cells.Tumor suppressor p53 stole the AKT in hypoxia.Hypoxic stress facilitates acute activation and chronic downregulation of fanconi anemia proteins.Anti-tumor Activity of miniPEG-γ-Modified PNAs to Inhibit MicroRNA-210 for Cancer Therapy.A cell-penetrating antibody inhibits human RAD51 via direct binding.Suppression of homology-dependent DNA double-strand break repair induces PARP inhibitor sensitivity in VHL-deficient human renal cell carcinoma.Peptide Nucleic Acids as a Tool for Site-Specific Gene Editing.The hypoxic tumor microenvironment in vivo selects the cancer stem cell fate of breast cancer cells.In utero nanoparticle delivery for site-specific genome editing.Pathological Oxidation of PTPN12 Underlies ABL1 Phosphorylation in Hereditary Leiomyomatosis and Renal Cell CarcinomaHigh-throughput Evaluation of Protein Migration and Localization after Laser Micro-IrradiationPTEN Regulates Nonhomologous End Joining By Epigenetic Induction of NHEJ1/XLFHypoxia Promotes Resistance to EGFR Inhibition in NSCLC Cells via the Histone Demethylases, LSD1 and PLU-1Peptide Nucleic Acids and Gene Editing: Perspectives on Structure and RepairKu80-targeted pH-sensitive peptide-PNA conjugates are tumor selective and sensitize cancer cells to ionizing radiationMcp1 Promotes Macrophage-Dependent Cyst Expansion in Autosomal Dominant Polycystic Kidney DiseaseCediranib suppresses homology-directed DNA repair through down-regulation of BRCA1/2 and RAD51Electron-Mediated Aminyl and Iminyl Radicals from C5 Azido-Modified Pyrimidine Nucleosides Augment Radiation Damage to Cancer Cells
P50
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P50
description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Peter M Glazer
@en
Peter M Glazer
@nl
type
label
Peter M Glazer
@en
Peter M Glazer
@nl
prefLabel
Peter M Glazer
@en
Peter M Glazer
@nl
P31
P496
0000-0003-4525-5560