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Electrophysiological Properties and Viability of Neonatal Rat Ventricular Myocyte Cultures with Inducible ChR2 ExpressionImpaired mitochondrial network excitability in failing guinea-pig cardiomyocytesInhibiting Na+/K+ ATPase can impair mitochondrial energetics and induce abnormal Ca2+ cycling and automaticity in guinea pig cardiomyocytes.Cardiomyocyte mitochondrial oxidative stress and cytoskeletal breakdown in the heart with a primary volume overload.Targeted biopharmaceuticals for cancer treatment.Knockout of the ATPase inhibitory factor 1 protects the heart from pressure overload-induced cardiac hypertrophy.Comparative proteomics analysis of high n-butanol producing metabolically engineered Clostridium tyrobutyricum.
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Q29994747-E3B41D9B-E7C3-4321-A406-8A7A9BD2A3CFQ30692654-27E4831E-37DC-474C-A973-39D78318CD93Q35145345-45C0B838-0A4D-422F-8764-CFD0C101594DQ35178659-A32DE504-4196-4931-8193-EC69295C9337Q38228647-E30F4560-89E2-40F0-BEA2-2A1D28AC9ECDQ41621006-0F886938-9CE5-49D8-8819-0A7A3465C725Q46807028-23949662-655B-470F-87CE-BC7D01F8FBB4
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description
investigador
@es
researcher
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name
Lufang Zhou
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type
label
Lufang Zhou
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prefLabel
Lufang Zhou
@en
P31
P496
0000-0002-1321-8442