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Crystal Structure of the Vibrio cholerae Quorum-Sensing Regulatory Protein HapRStructure of Vibrio cholerae ToxT reveals a mechanism for fatty acid regulation of virulence genesThe crystal structure of AphB, a virulence gene activator from Vibrio cholerae, reveals residues that influence its response to oxygen and pHH-NS binding and repression of the ctx promoter in Vibrio choleraeThe quorum sensing regulator HapR downregulates the expression of the virulence gene transcription factor AphA in Vibrio cholerae by antagonizing Lrp- and VpsR-mediated activation.Dual regulation of genes involved in acetoin biosynthesis and motility/biofilm formation by the virulence activator AphA and the acetate-responsive LysR-type regulator AlsR in Vibrio cholerae.Characterization of BreR interaction with the bile response promoters breAB and breR in Vibrio cholerae.Vibrio cholerae AphA uses a novel mechanism for virulence gene activation that involves interaction with the LysR-type regulator AphB at the tcpPH promoter.The LysR-type virulence activator AphB regulates the expression of genes in Vibrio cholerae in response to low pH and anaerobiosisThe virulence activator AphA links quorum sensing to pathogenesis and physiology in Vibrio cholerae by repressing the expression of a penicillin amidase gene on the small chromosomeBinding site requirements of the virulence gene regulator AphB: differential affinities for the Vibrio cholerae classical and El Tor tcpPH promoters.The alternative sigma factor sigma(E) plays an important role in intestinal survival and virulence in Vibrio cholerae.Parallel quorum sensing systems converge to regulate virulence in Vibrio cholerae.The 40-residue insertion in Vibrio cholerae FadR facilitates binding of an additional fatty acyl-CoA ligand.Proteolysis of virulence regulator ToxR is associated with entry of Vibrio cholerae into a dormant state.Integration host factor positively regulates virulence gene expression in Vibrio cholerae.Crystal structure of the virulence gene activator AphA from Vibrio cholerae reveals it is a novel member of the winged helix transcription factor superfamily.Identification of a Small Molecule Activator for AphB, a LysR-Type Virulence Transcriptional Regulator in Vibrio cholerae.Role of FlgT in anchoring the flagellum of Vibrio cholerae.The Fatty Acid Regulator FadR Influences the Expression of the Virulence Cascade in the El Tor Biotype of Vibrio cholerae by Modulating the Levels of ToxT via Two Different MechanismsLevels of the secreted Vibrio cholerae attachment factor GbpA are modulated by quorum-sensing-induced proteolysis.Requirements for Vibrio cholerae HapR binding and transcriptional repression at the hapR promoter are distinct from those at the aphA promoterRegulation of virulence gene expression in Vibrio cholerae by quorum sensing: HapR functions at the aphA promoter
P50
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P50
name
Karen Skorupski
@en
type
label
Karen Skorupski
@en
prefLabel
Karen Skorupski
@en