about
Histone H3K4 monomethylation catalyzed by Trr and mammalian COMPASS-like proteins at enhancers is dispensable for development and viability.An Mll4/COMPASS-Lsd1 epigenetic axis governs enhancer function and pluripotency transition in embryonic stem cells.PAF1 regulation of promoter-proximal pause release via enhancer activation.Impactful science as the guiding principle.Resetting the epigenetic balance of Polycomb and COMPASS function at enhancers for cancer therapyAberrant activation of non-coding RNA targets of transcriptional elongation complexes contributes to TDP-43 toxicityPromoter bivalency favors an open chromatin architecture in embryonic stem cellsTET2 coactivates gene expression through demethylation of enhancersAuthor Correction: Aberrant activation of non-coding RNA targets of transcriptional elongation complexes contributes to TDP-43 toxicityMetarrestin, a perinucleolar compartment inhibitor, effectively suppresses metastasis.LEDGF and HDGF2 relieve the nucleosome-induced barrier to transcription in differentiated cellsCATACOMB: An endogenous inducible gene that antagonizes H3K27 methylation activity of Polycomb repressive complex 2 via an H3K27M-like mechanismThe ATPase module of mammalian SWI/SNF family complexes mediates subcomplex identity and catalytic activity-independent genomic targetingβ-Catenin/Tcf7l2-dependent transcriptional regulation of GLUT1 gene expression by Zic family proteins in colon cancerUncoupling histone H3K4 trimethylation from developmental gene expression via an equilibrium of COMPASS, Polycomb and DNA methylationMedicine in the time of corona: Fundamental molecular research is “essential”
P50
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P50
description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Ali Shilatifard
@en
Ali Shilatifard
@nl
type
label
Ali Shilatifard
@en
Ali Shilatifard
@nl
prefLabel
Ali Shilatifard
@en
Ali Shilatifard
@nl
P31
P496
0000-0002-7490-2854