about
Ras oncogene-independent activation of RALB signaling is a targetable mechanism of escape from NRAS(V12) oncogene addiction in acute myeloid leukemia.Stat5 is critical for the development and maintenance of myeloproliferative neoplasm initiated by Nf1 deficiencyRALB provides critical survival signals downstream of Ras in acute myeloid leukemiaTargeting Ras signaling in AML: RALB is a small GTPase with big potential.SEMA4C is a novel target to limit osteosarcoma growth, progression, and metastasisAn Indole-Chalcone Inhibits Multidrug-Resistant Cancer Cell Growth by Targeting MicrotubulesHighly efficient multiplex human T cell engineering without double-strand breaks using Cas9 base editorsAuthor Correction: Highly efficient multiplex human T cell engineering without double-strand breaks using Cas9 base editors
P50
Q33779805-926D05E1-2E82-4BD0-A600-F06BE2F71F91Q37304113-BFB53A4C-C425-48B0-9F91-C4704F8E1CE4Q37662202-D57CD90E-7830-40E8-86C2-FC79CE2A382BQ48166242-02623D17-C32F-488A-AC6A-674008DE9492Q90468284-1471AE35-C6F1-49C2-8B02-C2C79827A0B9Q90494509-5EB302AD-05DE-4B9F-95A3-3E9A99C14833Q91344495-8CB59F4F-83FC-47BA-A925-DFF856D72C22Q91767739-CBF1211B-4CD5-42E1-B4BA-F3D4BB0E7900
P50
description
researcher
@en
wetenschapper
@nl
name
Emily J Pomeroy
@en
Emily J Pomeroy
@nl
type
label
Emily J Pomeroy
@en
Emily J Pomeroy
@nl
prefLabel
Emily J Pomeroy
@en
Emily J Pomeroy
@nl
P31
P496
0000-0001-7690-3570